Zobrazeno 1 - 10
of 28
pro vyhledávání: '"Deniz Kanca"'
Autor:
Burcu Çaykara, Bengü Tokat, Ender Coşkunpınar, Özlem Küçükhüseyin, Deniz Kanca Demirci, Zehra Buğra, Gülçin Özkara, Oğuz Öztürk, Sadrettin Pençe, Hülya Yılmaz Aydoğan
Publikováno v:
Türk Kardiyoloji Derneği Arşivi, Vol 49, Iss 8, Pp 641-653 (2021)
Externí odkaz:
https://doaj.org/article/85b83a114c5a40a89d25f763117a5538
Autor:
Sadiye Nur Dalgic, Hulya Yilmaz Aydogan, Oguz Ozturk, Sadrettin Pence, Deniz Kanca Demirci, Okay Abaci, Cuneyt Kocas, Yalcin Dalgic, Cem Bostan, Ahmet Yildiz
Publikováno v:
Journal of International Medical Research, Vol 48 (2020)
Objective Endothelin-1 (ET-1) promotes the progression and induction of sclerotic renal changes in end-stage kidney disease. Membrane-bound endothelin-converting enzyme 1 (ECE-1) is involved in the production of ET-1. The aim of this study was to ass
Externí odkaz:
https://doaj.org/article/e7ab0bdebd344ad4b34262883ba4191d
Akademický článek
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Autor:
Hülya Yılmaz Aydoğan, Sukran Poyrazoglu, Ilhan Satman, Feyza Darendeliler, Kazim Yalcin Arga, Oğuz Öztürk, Gizem Gulfidan, Nurdan Gul, Deniz Kanca Demirci, Canan Cacina, Aslı Derya Kardelen Al, Yildiz Tutuncu
Publikováno v:
OMICS: A Journal of Integrative Biology. 25:431-449
Diabetes is a common disorder with a heterogeneous clinical presentation and an enormous burden on health care worldwide. About 1-6% of patients with diabetes suffer from maturity-onset diabetes of the young (MODY), the most common form of monogenic
Autor:
AYCA DIREN, DENIZ KANCA DEMIRCI, NURDAN GUL, BURCIN KARACANLI, AYKUT BAYKUT, YILDIZ TUTUNCU, OGUZ OZTURK, ILHAN SATMAN, HULYA YILMAZ-AYDOGAN
Publikováno v:
In Vivo
Background/aim: high-sensitivity C-reactive protein (hs-CRP) is used in the differential diagnosis of maturity-onset diabetes of the young (MODY)-3, but other inflammatory markers have not been investigated in MODY patients. We aimed to compare the s
Autor:
Hulya Yilmaz Aydogan, Nurdan Gul, Deniz Kanca Demirci, Ummu Mutlu, Gizem Gulfidan, Kazim Yalcin Arga, Aclan Ozder, Ahmet Adil Camli, Yildiz Tutuncu, Oguz Ozturk, Canan Cacina, Feyza Darendeliler, Sukran Poyrazoglu, Ilhan Satman
Maturity-onset diabetes of the young (MODY) is a highly heterogeneous group of monogenic and nonautoimmune diseases. Misdiagnosis of MODY is a widespread problem and about 5% of patients with type 2 diabetes mellitus and nearly 10% with type 1 diabet
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f27631351930d23f109b9a15da374ffd
http://hdl.handle.net/20.500.12645/30482
http://hdl.handle.net/20.500.12645/30482
Akademický článek
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Publikováno v:
Volume: 4, Issue: 1 41-68
Haliç Üniversitesi Fen Bilimleri Dergisi
Haliç Üniversitesi Fen Bilimleri Dergisi
Gençlerin Erişkin Başlangıçlı Diyabeti (MODY) otozomal dominant kalıtım ile karakterize monogenik bir hastalıktır. Şimdiye kadar 14 farklı gende çok sayıda heterozigot mutasyonlar tanımlanmış olup bu mutasyonların dağılımı her
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ce7659c3fd2f8605f5665c66aa5daab5
https://dergipark.org.tr/tr/pub/hafebid/issue/61135/880832
https://dergipark.org.tr/tr/pub/hafebid/issue/61135/880832
Autor:
Demirci, Deniz Kanca, Darendeliler, Feyza, Poyrazoglu, Sukran, Al, Asli Derya Kardelen, Gul, Nurdan, Tutuncu, Yildiz, Gulfidan, Gizem, Arga, Kazim Yalcin, Cacina, Canan, Ozturk, Oguz, Aydogan, Hulya Yilmaz, Satman, Ilhan
Diabetes is a common disorder with a heterogeneous clinical presentation and an enormous burden on health care worldwide. About 1-6% of patients with diabetes suffer from maturity-onset diabetes of the young (MODY), the most common form of monogenic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=r39c86a4b39b::14452b459da938ecc0787331f2710861
https://aperta.ulakbim.gov.tr/record/230806
https://aperta.ulakbim.gov.tr/record/230806
Autor:
Nurdan Gul, Bengu Tokat, Deniz Kanca-Demirci, Ozlem Kucukhuseyin, Hulya Yilmaz-Aydogan, Oğuz Öztürk, Ilhan Satman, Aclan Ozder
Publikováno v:
Gene. 763
Background: The nuclear receptors Rev-erb alpha and Rev-erb beta are transcription factors that regulate the function of genes in glucose and lipid metabolism, and they also form a link between circadian rhythm and metabolism. We evaluated the variat