Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Deniz B Dedeoglu"'
Publikováno v:
Acta Cardiologica. 67:87-96
The aim of this study is to compare possible protective effects of zofenopril, enalapril and valsartan against both ischaemia/reperfusion injury as well as acute doxorubicin cardiotoxicity. All three agents have never been compared in this setting be
Autor:
Kyoji Urayama, Nadia Messaddeq, Gulen Turkeri, Canan G. Nebigil, Deniz B. Dedeoglu, Hitoshi Kurose, Célia Guilini
Publikováno v:
AJP-Heart and Circulatory Physiology
AJP-Heart and Circulatory Physiology, American Physiological Society, 2010, 298 (3), pp.H844-H852. ⟨10.1152/ajpheart.00898.2009⟩
AJP-Heart and Circulatory Physiology, American Physiological Society, 2010, 298 (3), pp.H844-H852. ⟨10.1152/ajpheart.00898.2009⟩
Prokineticins are secreted peptides that activate two G protein-coupled receptors: PKR1 and PKR2. Prokineticins induce angiogenesis and fenestration, but the cognate receptors involved in these functions are unknown. We hypothesized a role for prokin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cdb397ff565823498d1b25b35f63d80a
https://hal.archives-ouvertes.fr/hal-02391089
https://hal.archives-ouvertes.fr/hal-02391089
Autor:
Stefan Frantz, Kyoji Urayama, Deniz B. Dedeoglu, Canan G. Nebigil, Nadia Messaddeq, Georg Ertl, Célia Guilini
Publikováno v:
Cardiovascular Research
Cardiovascular Research, Oxford University Press (OUP), 2009, 81 (1), pp.28-37. ⟨10.1093/cvr/cvn251⟩
Cardiovascular Research, Oxford University Press (OUP), 2008, 81 (1), pp.28-37. ⟨10.1093/cvr/cvn251⟩
Cardiovascular Research, 2009, 81 (1), pp.28-37. ⟨10.1093/cvr/cvn251⟩
Cardiovascular Research, Oxford University Press (OUP), 2009, 81 (1), pp.28-37. ⟨10.1093/cvr/cvn251⟩
Cardiovascular Research, Oxford University Press (OUP), 2008, 81 (1), pp.28-37. ⟨10.1093/cvr/cvn251⟩
Cardiovascular Research, 2009, 81 (1), pp.28-37. ⟨10.1093/cvr/cvn251⟩
International audience; AIMS: Prokineticins are small secreted bioactive molecules. They exert their biological activity by binding to two G protein-coupled receptors. Previously, we have shown that the overexpression of prokineticin receptor-1 (PKR1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ffcb55c57018cd9c1aab4e984f8c47f3
https://www.hal.inserm.fr/inserm-00370169
https://www.hal.inserm.fr/inserm-00370169
Autor:
Kyoji Urayama, Deniz B. Dedeoglu, Célia Guilini, Stefan Frantz, Georg Ertl, Nadia Messaddeq, Canan G. Nebigil
Publikováno v:
Cardiovascular Research; Jan2009, Vol. 81 Issue 1, p28-37, 10p