Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Denise Harhausen"'
Autor:
Denise Harhausen, Violetta Sudmann, Uldus Khojasteh, Jochen Müller, Marietta Zille, Keith Graham, Andrea Thiele, Thomas Dyrks, Ulrich Dirnagl, Andreas Wunder
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e69529 (2013)
Inflammation is a pathophysiological hallmark of many diseases of the brain. Specific imaging of cells and molecules that contribute to cerebral inflammation is therefore highly desirable, both for research and in clinical application. The 18 kDa tra
Externí odkaz:
https://doaj.org/article/458fd263d1704dd48e33e163ae831af5
Autor:
Yoash Chassidim, Eyk Schellenberger, Uwe Heinemann, Richard Kovács, Ofer Prager, Lyn Kamintsky, Marietta Zille, Alon Friedman, Lena Figge, Denise Harhausen, Karl Schoknecht, Udi Vazana
Publikováno v:
Journal of Cerebral Blood Flow & Metabolism. 34:1791-1801
Focal cerebral ischemia is among the main causes of death and disability worldwide. The ischemic core often progresses, invading the peri-ischemic brain; however, assessing the propensity of the peri-ischemic brain to undergo secondary damage, unders
Autor:
Eyk Schellenberger, Jens Steinbrink, Peyman Bahmani, Ryan Cordell, Leonard Hofstra, Chris P. M. Reutelingsperger, Jochen Müller, Marietta Zille, Denise Harhausen, Andreas Wunder, Tracy D. Farr, Jan Klohs, Ulrich Dirnagl
Publikováno v:
Journal of Cerebral Blood Flow & Metabolism; Vol 31
Journal of Cerebral Blood Flow and Metabolism, 31(5), 1311-1320. SAGE Publications Inc.
Journal of Cerebral Blood Flow & Metabolism
Journal of Cerebral Blood Flow and Metabolism, 31(5), 1311-1320. SAGE Publications Inc.
Journal of Cerebral Blood Flow & Metabolism
To monitor stroke-induced brain damage and assess neuroprotective therapies, specific imaging of cell death after cerebral ischemia in a noninvasive manner is highly desirable. Annexin A5 has been suggested as a marker for imaging cell death under va
Autor:
Philip F. Stahel, Marina Botto, Karen Gertz, Hans Lehrach, Vincent Prinz, Denise Harhausen, George Trendelenburg, Philippe Gasque, Gina Ziegler, Matthias Endres, Wilfried Nietfeld, Ulrich Dirnagl
Publikováno v:
Journal of Immunology
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2010, 184 (11), pp.6407--6417. ⟨10.4049/jimmunol.0902342⟩
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2010, 184 (11), pp.6407--6417. ⟨10.4049/jimmunol.0902342⟩
The stem-cell marker CD93 (AA4.1/C1qRp) has been described as a potential complement C1q-receptor. Its exact molecular function, however, remains unknown. By using global expression profiling we showed that CD93-mRNA is highly induced after transient
Autor:
Christina Röhr, Gina Ziegler, George Trendelenburg, Vincent Prinz, Claudia Schepers, Hans Lehrach, Wilfried Nietfeld, Denise Harhausen, Janett König, Olaf Hoffmann
Publikováno v:
Biochemical and Biophysical Research Communications. 359:574-579
A significant up-regulation of Toll-like-receptor (TLR) mRNAs between 3 and 48 h reperfusion time after induction of transient focal cerebral ischemia for 1h was revealed by applying global gene expression profiling in postischemic mouse brains. Comp
Autor:
Marietta Zille, Andreas Wunder, Chris P. M. Reutelingsperger, Ulrich Dirnagl, Denise Harhausen, Marijke De Saint-Hubert, Roger Michel
Publikováno v:
Journal of Cerebral Blood Flow and Metabolism, 34(9), 1568-1570. SAGE Publications Inc.
Journal of cerebral blood flow & metabolism 34(9), e1-e7 (2014). doi:10.1038/jcbfm.2014.115
Scopus-Elsevier
Journal of cerebral blood flow & metabolism 34(9), e1-e7 (2014). doi:10.1038/jcbfm.2014.115
Scopus-Elsevier
Cell death is one of the pathophysiological hallmarks after stroke. Markers to image cell death pathways in vivo are highly desirable. We previously showed that fluorescently labeled Annexin A5 (An×A5), which binds specifically to phosphatidylserine
Autor:
Kathrin Baldauf, Jenni Neubert, Henning Scheich, Ulrich Dirnagl, Klaus G. Reymann, Andreas Wunder, Ulrich H. Schröder, Franziska Stöber, Marietta Zille, Denise Harhausen, Iryna Ziabreva, Jürgen Goldschmidt
Publikováno v:
Journal of Cerebral Blood Flow & Metabolism
Journal of Cerebral Blood Flow & Metabolism; Vol 34
Journal of cerebral blood flow & metabolism 34(1), 144-152 (2013). doi:10.1038/jcbfm.2013.177
Journal of Cerebral Blood Flow & Metabolism; Vol 34
Journal of cerebral blood flow & metabolism 34(1), 144-152 (2013). doi:10.1038/jcbfm.2013.177
Neuronal damage shortly after onset or after brief episodes of cerebral ischemia has remained difficult to assess with clinical and preclinical imaging techniques as well as with microscopical methods. We here show, in rodent models of middle cerebra
Autor:
Andrea Thiele, Ulrich Dirnagl, Andreas Wunder, Keith Graham, Uldus Khojasteh, Denise Harhausen, Thomas Dyrks, Violetta Sudmann, Marietta Zille, Jochen Müller
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e69529 (2013)
PLoS ONE; Vol 8
PLoS ONE
PLoS ONE; Vol 8
PLoS ONE
Inflammation is a pathophysiological hallmark of many diseases of the brain. Specific imaging of cells and molecules that contribute to cerebral inflammation is therefore highly desirable, both for research and in clinical application. The 18 kDa tra
Autor:
Dorette Freyer, George Trendelenburg, Uldus Khojasteh, Wilfried Nietfeld, Denise Harhausen, Gina Ziegler
Publikováno v:
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 31(2)
Reduced infarct volume in TLR2-knockout mice compared with C57Bl/6 wild-type mice has recently been shown in experimental stroke and confirmed in this study. We now also show a significant decrease of CD11b-positive cell counts and decreased neuronal
Autor:
Denise Harhausen, Uldus Khojasteh, Wilfried Nietfeld, Philip F. Stahel, Ulrich Dirnagl, George Trendelenburg, B. Paul Morgan
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 7, Iss 1, p 15 (2010)
Journal of Neuroinflammation, Vol 7, Iss 1, p 15 (2010)
Background The complement system is a crucial mediator of inflammation and cell lysis after cerebral ischemia. However, there is little information about the exact contribution of the membrane attack complex (MAC) and its inhibitor-protein CD59. Meth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b8dc0973d631e30e25cbdabc68d883f