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pro vyhledávání: '"Denil, Simon LIJ"'
Autor:
Chia, Crystal Y, Madrigal, Pedro, Denil, Simon LIJ, Martinez, Iker, Garcia-Bernardo, Jose, El-Khairi, Ranna, Chhatriwala, Mariya, Shepherd, Maggie H, Hattersley, Andrew T, Dunn, N Ray, Vallier, Ludovic
Publikováno v:
Stem Cell Reports, Vol 12, Iss 1, Pp 57-70 (2019)
Summary: Heterozygous de novo mutations in GATA6 are the most frequent cause of pancreatic agenesis in humans. In mice, however, a similar phenotype requires the biallelic loss of Gata6 and its paralog Gata4. To elaborate the human-specific requireme
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::0f9bda69da5d359bd752517d57e35c86
http://hdl.handle.net/10220/49231
http://hdl.handle.net/10220/49231
Autor:
Chia, Crystal Y, Madrigal, Pedro, Denil, Simon LIJ, Martinez, Iker, Garcia-Bernardo, Jose, El-Khairi, Ranna, Chhatriwala, Mariya, Shepherd, Maggie H, Hattersley, Andrew T, Dunn, N Ray, Vallier, Ludovic
Heterozygous de novo mutations in GATA6 are the most frequent cause of pancreatic agenesis in humans. In mice, however, a similar phenotype requires the biallelic loss of Gata6 and its paralog Gata4. To elaborate the human-specific requirements for G
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f912f882c0df320aa6df520b9962f223