Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Dena Arizanovska"'
Publikováno v:
Cell Death & Differentiation. 30:397-406
Hallmark pathological features of brain trauma are axonal degeneration and demyelination because myelin-producing oligodendrocytes (OLs) are particularly vulnerable to injury-induced death signals. To reveal mechanisms responsible for this OL loss, w
Autor:
Stephen A. Tapanes, Dena Arizanovska, Madelen M. Díaz, Oluwarotimi O. Folorunso, Theresa Harvey, Stephanie E. Brown, Inna Radzishevsky, Liesl N. Close, Jonathan R. Jagid, Joacir Graciolli Cordeiro, Herman Wolosker, Darrick T. Balu, Daniel J. Liebl
Publikováno v:
Glia. 70:1133-1152
Synaptic damage is one of the most prevalent pathophysiological responses to traumatic CNS injury and underlies much of the associated cognitive dysfunction; however, it is poorly understood. The D-amino acid, D-serine, serves as the primary co-agoni
Publikováno v:
PsychopharmacologyReferences. 239(10)
Drug-induced potentiation of ventral tegmental area (VTA) glutamate signaling contributes critically to the induction of sensitization - an enhancement in responding to a drug following exposure which is thought to reflect neural changes underlying d
Publikováno v:
Volume 15, Issue 1. 15
Syndecan (Sdc) is a transmembrane heparan sulfate proteoglycan that plays a crucial role in axon guidance and synapse formation during CNS development in Drosophila melanogaster. To further examine the effect of syndecan on CNS function, Sdc23 mutant