Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Delai Xu"'
Publikováno v:
Heliyon, Vol 10, Iss 10, Pp e30903- (2024)
Rheumatoid arthritis (RA) is a persistent autoimmune condition with no identified cure currently. Recently, scientists have applied metabolomics to investigate altered metabolic profiles and unique diseases-associated metabolic signatures. Herein, we
Externí odkaz:
https://doaj.org/article/68a7c43b08f84b38b238b3556390dfe6
Autor:
Yuchen Qu, Cunjin Su, Qinhong Zhao, Aiming Shi, Fenglun Zhao, Liuxing Tang, Delai Xu, Zheng Xiang, Yang Wang, Yueyuan Wang, Jie Pan, Yunli Yu
Publikováno v:
Frontiers in Pharmacology, Vol 13 (2022)
A growing body of evidence suggests that gut microbiota could participate in the progression of depression via the microbiota–gut–brain axis. However, the detailed microbial metabolic profile changes in the progression of depression is still not
Externí odkaz:
https://doaj.org/article/3b1083dc86af43419dde92d2ba3ae35c
Publikováno v:
IEEE Transactions on Systems, Man, and Cybernetics: Systems. :1-9
Advances and Application of a Novel Oral Anticoagulant in Specific Populations: Dabigatran Etexilate
Publikováno v:
Current Drug Metabolism. 21:106-111
Background: Dabigatran etexilate (DE) was approved by the FDA in 2010 to reduce the risk of stroke and systemic embolism in adults with Non-valvular Atrial Fibrillation (NVAF). Compared with warfarin, a traditional anticoagulant drug, DE exhibits a s
Autor:
Qifeng Ding, Zeng-li Zhang, Zhonghua Xu, Bin Qian, Yufei Xing, Minhua Shi, Zheng Ying, Xue Pan, Delai Xu, Anyuan Zhong, Caicun Zhou, Tong Zhou, Hongfeng Wang, Chenghong Ji
Publikováno v:
Biomedicine & Pharmacotherapy. 89:1055-1060
The cellular protein degradation system, such as proteasomal or autophagy-lysosomal system plays an important role in the pathogenesis of a variety of human diseases including cancer. Transcription factor EB (TFEB) is a master transcriptional factor
Publikováno v:
Autophagy. 12:707-708
In a recent paper we addressed the mechanism by which defective autophagy contributes to TARDBP/TDP-43-mediated neurodegenerative disorders. We demonstrated that TARDBP regulates MTORC1-TFEB signaling by targeting RPTOR/raptor, a key component and an