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Cardiolipin (CL) is a phospholipid required for proper mitochondrial function. Tafazzin remodels CL to create highly unsaturated fatty acid chains. However, when tafazzin is mutated, CL remodeling is impeded, leading to mitochondrial dysfunction and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::75ccf47abc5e2a8f7d8dadaad05c59bd
https://doi.org/10.1101/2022.01.06.475267
https://doi.org/10.1101/2022.01.06.475267