Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Dedipya, Yelamanchili"'
Autor:
Dedipya Yelamanchili, Baiba K. Gillard, Antonio M. Gotto, Jr., Miguel Caínzos Achirica, Khurram Nasir, Alan T. Remaley, Corina Rosales, Henry J. Pownall
Publikováno v:
Journal of Lipid Research, Vol 66, Iss 1, Pp 100707- (2025)
High-density lipoprotein (HDL)-free cholesterol (FC) transfers to other lipoproteins and cells, the former by a spontaneous mechanism and the latter by both spontaneous and receptor-mediated mechanisms. Macrophages are an important cell type in all s
Externí odkaz:
https://doaj.org/article/6d7cba0aea5d4f68b28b03eccc94afe4
Autor:
Ziyi Wang, Dedipya Yelamanchili, Jing Liu, Antonio M. Gotto, Jr., Corina Rosales, Baiba K. Gillard, Henry J. Pownall
Publikováno v:
Journal of Lipid Research, Vol 64, Iss 11, Pp 100456- (2023)
Compared with WT mice, HDL receptor-deficient (Scarb1−/−) mice have higher plasma levels of free cholesterol (FC)-rich HDL and exhibit multiple pathologies associated with a high mol% FC in ovaries, platelets, and erythrocytes, which are reversed
Externí odkaz:
https://doaj.org/article/9ab64245fa4046f68267f451fcd9985d
Autor:
Corina Rosales, Dedipya Yelamanchili, Baiba K. Gillard, Jing Liu, Antonio M. Gotto, Jr., Henry J. Pownall
Publikováno v:
Journal of Lipid Research, Vol 64, Iss 2, Pp 100327- (2023)
Human female infertility, 20% of which is idiopathic, is a public health problem for which better diagnostics and therapeutics are needed. A novel cause of infertility emerged from studies of female mice deficient in the HDL receptor gene (Scarb1). T
Externí odkaz:
https://doaj.org/article/60746f5620db47cc9e142c6c9b7a6e52
Autor:
Corina, Rosales, Dedipya, Yelamanchili, Baiba K, Gillard, Jing, Liu, Antonio M, Gotto, Henry J, Pownall
Publikováno v:
Journal of lipid research.
Human female infertility, 20% of which is idiopathic, is a public health problem for which better diagnostics and therapeutics are needed. A novel cause of infertility emerged from studies of female mice deficient in the HDL receptor gene (Scarb1). T
Autor:
Baiba K Gillard, Ziyi Wang, Jing Liu, Dedipya Yelamanchili, Antonio M Gotto, Corina Rosales, Henry J Pownall
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 42
Aim: In humans, very high plasma HDL-cholesterol concentrations are associated with increased all cause- and atherosclerotic cardiovascular disease (ASCVD)-mortality. The HDL receptor-deficient mouse (Scarb1 -/- ) is a robust model of this phenotype
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 42
Although plasma HDL-C levels negatively correlate with atherosclerotic cardiovascular disease (ACVD), attempts to reduce ACVD risk by raising plasma HDL have disappointed. Thus, hypotheses about salutary HDL effects have shifted from higher-is-better
Autor:
Jing Liu, Baiba K Gillard, Dedipya Yelamanchili, Ziyi Wang, Antonio M Gotto, Corina Rosales, Henry J Pownall
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 41
Aim: In humans, very high plasma HDL-cholesterol concentrations are associated with increased all cause- and atherosclerotic cardiovascular disease (ASCVD)-mortality. The HDL receptor-deficient mouse (Scarb1 -/- ), a robust model of this phenotype, i
Autor:
Antonio M. Gotto, Corina Rosales, Dedipya Yelamanchili, Jing Liu, Baiba K. Gillard, Henry J. Pownall
Publikováno v:
Arterioscler Thromb Vasc Biol
Objective: Overall and atherosclerosis-associated mortality is elevated in humans with very high HDL (high-density lipoprotein) cholesterol concentrations. Mice with a deficiency of the HDL receptor, Scarb1 (scavenger receptor class B type 1), are a
Autor:
Z. Wang, Dedipya Yelamanchili, Baiba K. Gillard, Antonio M. Gotto, John R. Barr, J. Rees, Henry J. Pownall, B. Qi, Z. Kuklenyik, Corina Rosales
Publikováno v:
Atherosclerosis. 331:e126-e127
Autor:
Baiba K. Gillard, Dedipya Yelamanchili, Corina Rosales, Antonio M. Gotto, Ayrea Hurley, W. Sean Davidson, Henry J. Pownall, Jing Liu, Willam R. Lagor
Publikováno v:
Biochimica et biophysica acta. Molecular and cell biology of lipids. 1865(12)
Objective Apolipoprotein A1 (APOA1) is essential to reverse cholesterol transport, a physiologically important process that protects against atherosclerotic cardiovascular disease. APOA1 is a 28 kDa protein comprising multiple lipid-binding amphiphat