Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Debra Salvi"'
Autor:
Mark Pilkinton, Jiaping Xue, Debra Salvi, Hiroaki Kiyokawa, Oscar R. Colamonici, Raudel Sandoval
Publikováno v:
Journal of Biological Chemistry. 279:32275-32280
The regulation of cell growth is one of the most important effects of type I interferons (IFNs). This response may involve a cytostatic effect or the induction of apoptosis depending on the cell context. Often the growth-inhibitory response of type I
Autor:
Anna Usacheva, Oscar R. Colamonici, Debra Salvi, David E. Levy, Xinyong Tian, Raudel Sandoval
Publikováno v:
The Journal of Immunology. 171:2989-2994
The WD repeat-containing protein receptor for activated protein kinase C (RACK)-1 has been linked to a variety of signaling systems including protein kinase C, growth factors, and IFNs. In the IFN system, RACK-1 functions as an adaptor recruiting the
Autor:
Tamara E. Levin, Judith A. Badner, Katherine A. Walczyk, Bryan J. Mowry, Saitou Naruya, Qiuhe Cao, Douglas F. Levinson, Raymond R. Crowe, Jennifer M. Taylor, Amado Del Rosario, Debra Salvi, Alan R. Sanders, Jeremy M. Silverman, Josep M. Comeron, Pablo V. Gejman, Anibal Cravchik
Publikováno v:
Genomics. 72:1-14
We systematically and comprehensively investigated polymorphisms of the HTR1B gene as well as their linkage disequilibrium and ancestral relationships. We have detected the following polymorphisms in our sample via denaturing gradient gel electrophor
Autor:
Raudel, Sandoval, Jiaping, Xue, Mark, Pilkinton, Debra, Salvi, Hiroaki, Kiyokawa, Oscar R, Colamonici
Publikováno v:
The Journal of biological chemistry. 279(31)
The regulation of cell growth is one of the most important effects of type I interferons (IFNs). This response may involve a cytostatic effect or the induction of apoptosis depending on the cell context. Often the growth-inhibitory response of type I
Publikováno v:
Kidney International. (4):1063-1069
Selectivity of endotoxin-induced defect in endothelial calcium mobilization. Background We hypothesized that endotoxin (LPS) would impair bradykinin (BK)-induced calcium (Ca 2+ ) mobilization in aortic endothelial cells, perhaps due to cytotoxicity o