Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Deborah M. Lenda"'
Publikováno v:
The Journal of Immunology. 170:3915-3925
Autoimmune disease in MRL-Faslpr mice is characterized by fatal nephritis, systemic pathology, and autoantibodies, mimicking human lupus. We previously reported that 1) intrarenal IL-12 elicits nephritis by fostering the accumulation of intrarenal IF
Autor:
Deborah M Lenda, Matthew A. Boegehold
Publikováno v:
Journal of Vascular Research. 39:41-50
High dietary salt intake decreases the endothelium-dependent dilation of skeletal muscle arterioles by inhibiting local nitric oxide (NO) activity without changing vascular smooth muscle responsiveness to NO. Under these conditions, microvascular wal
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 279:H7-H14
In normotensive rats, an increase in dietary salt leads to decreased arteriolar responsiveness to acetylcholine (ACh) because of suppressed local nitric oxide (NO) activity. We evaluated the possibility that generation of reactive oxygen species in t
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 173(7)
Inflammation in the kidney and other tissues (lung, and salivary and lacrimal glands) is characteristic of MRL-Faslpr mice with features of lupus. Macrophages (Mφ) are prominent in these tissues. Given that 1) Mφ survival, recruitment, proliferatio
Autor:
Matthew A. Boegehold, Deborah M Lenda
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 282(2)
Increased salt intake attenuates the endothelium-dependent dilation of skeletal muscle arterioles by abolishing local nitric oxide (NO) activity. There is evidence of oxidative stress in arteriolar and venular walls of rats fed a high-salt diet, and