Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Dayi Qin"'
Publikováno v:
Cardiovascular Research. 52:246-254
Objective: The hypothesis being tested in the present study is that increased anisotropic properties occurs in the remodeled post-infarction heart due to spatial alterations in Kv channels expression and K+ currents of the remodeled myocardium. Metho
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b74efb35c143a98c000d56124c33158f
https://doi.org/10.1016/s0008-6363(01)00378-9
https://doi.org/10.1016/s0008-6363(01)00378-9
Publikováno v:
Journal of Cardiovascular Electrophysiology. 12:1055-1061
Calcineurin Inhibition and Postinfarction Remodeling.Introduction: After myocardial infarction (MI), the heart undergoes an adaptive remodeling process characterized by hypertrophy of the noninfarcted myocardium. Calcineurin, a Ca2+– calmodulin-reg
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba66231e21b6f9256b7d0b886f061975
https://doi.org/10.1046/j.1540-8167.2001.01055.x
https://doi.org/10.1046/j.1540-8167.2001.01055.x
Publikováno v:
Journal of Cardiovascular Electrophysiology. 12:218-225
Postinfarction Changes in Cardiac Na+ Channel.Introduction: After a myocardial infarction (MI), the heart undergoes a remodeling process that includes hypertrophy of noninfarcted left ventricular myocytes. Alterations in the genetic expression, inclu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2b8cb1ca8e0de402bc633126f6b46a9
https://doi.org/10.1046/j.1540-8167.2001.00218.x
https://doi.org/10.1046/j.1540-8167.2001.00218.x
Reexpression of T-type Ca2+ channel gene and current in post-infarction remodeled rat left ventricle
Publikováno v:
Cardiovascular Research. 46:442-449
Objective: T-type Ca2+ currents ( I Ca-T) are present in neonatal rat myocytes but is not detected in adult ventricular myocytes. The present study was designed to investigate the expression of the T-type Ca2+ channel gene and current in post-infarct
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::45e9368e31bc2f1f495ba12068682448
https://doi.org/10.1016/s0008-6363(00)00017-1
https://doi.org/10.1016/s0008-6363(00)00017-1
Publikováno v:
Circulation Research. 79:461-473
After myocardial infarction (MI), the noninfarcted myocardium undergoes significant hypertrophy as part of the post-MI structural remodeling. Electrophysiological changes associated with the hypertrophied remodeled myocardium may play a key role in a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67bd5ecd688c1d9dba0321733564e360
https://doi.org/10.1161/01.res.79.3.461
https://doi.org/10.1161/01.res.79.3.461
Publikováno v:
The Japanese Journal of Physiology. 41:333-339
The Ca(2+)-activated K+ channel was activated by a step increase in the intracellular Ca2+ concentration ([Ca2+]i) using the oil-gate concentration jump method in hippocampal neurons of rat. Limitations due to the unstirred volume between the excised
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2fabfcb52585437e1b6b84f8cd53d5a6
https://doi.org/10.2170/jjphysiol.41.333
https://doi.org/10.2170/jjphysiol.41.333
Publikováno v:
Circulation research. 85(9)
Abstract —Three weeks after myocardial infarction (MI) in the rat, remodeled hypertrophy of noninfarcted myocardium is at its maximum and the heart is in a compensated stage with no evidence of heart failure. Our hemodynamic measurements at this st
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::189b4b349712de750eff814caa8c28bc
https://pubmed.ncbi.nlm.nih.gov/10532953
https://pubmed.ncbi.nlm.nih.gov/10532953
Publikováno v:
The American journal of physiology. 258(1 Pt 2)
ATP-dependent decay and recovery of the inward rectifier and ATP-sensitive K+ channels were investigated using inside-out patch recording in cardiac myocytes. The solution facing the inner side of the membrane was instantaneously changed with the oil
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::010fb80f5e6aae333b4f7e13029ee583
https://pubmed.ncbi.nlm.nih.gov/2301614
https://pubmed.ncbi.nlm.nih.gov/2301614
Publikováno v:
Journal of the American College of Cardiology. 27:209
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dce897df1cef916b5a32699f43ab9689
https://doi.org/10.1016/s0735-1097(96)81700-1
https://doi.org/10.1016/s0735-1097(96)81700-1
Publikováno v:
American Journal of Physiology: Heart & Circulatory Physiology; 1990, Vol. 258 Issue 1, pH45-H50, 6p