Zobrazeno 1 - 10
of 25
pro vyhledávání: '"David M. Bland"'
Publikováno v:
PLoS Biology, Vol 22, Iss 5 (2024)
Externí odkaz:
https://doaj.org/article/ce4a96b898c646b5a5e0c9bb78d00a6b
Publikováno v:
Parasites & Vectors, Vol 14, Iss 1, Pp 1-15 (2021)
Abstract Background The human flea, Pulex irritans, is widespread globally and has a long association with humans, one of its principal hosts. Its role in plague transmission is still under discussion, although its high prevalence in plague-endemic r
Externí odkaz:
https://doaj.org/article/82484b2637ba42ae9973e92256b3afd3
Publikováno v:
Biomolecules, Vol 11, Iss 2, p 210 (2021)
The ability to cause plague in mammals represents only half of the life history of Yersinia pestis. It is also able to colonize and produce a transmissible infection in the digestive tract of the flea, its insect host. Parallel to studies of the mole
Externí odkaz:
https://doaj.org/article/c90a57d5f30143d4a38cd4d7a3f9f012
Autor:
David M. Bland, Deborah M. Anderson
Publikováno v:
mBio, Vol 4, Iss 6 (2013)
ABSTRACT Vector-borne infections begin in the dermis when a pathogen is introduced by an arthropod during a blood meal. Several barriers separate an invading pathogen from its replicative niche, including phagocytic cells in the dermis that activate
Externí odkaz:
https://doaj.org/article/1d6d40a7cdf44cef8ca7996fd280d199
Autor:
Adélaïde Miarinjara, David A Eads, David M Bland, Marc R Matchett, Dean E Biggins, B Joseph Hinnebusch
Publikováno v:
J Med Entomol
Prairie dogs in the western United States experience periodic epizootics of plague, caused by the flea-borne bacterial pathogen Yersinia pestis. An early study indicated that Oropsylla hirsuta (Baker), often the most abundant prairie dog flea vector
Publikováno v:
PLoS Pathogens, Vol 14, Iss 1, p e1006859 (2018)
Fleas can transmit Yersinia pestis by two mechanisms, early-phase transmission (EPT) and biofilm-dependent transmission (BDT). Transmission efficiency varies among flea species and the results from different studies have not always been consistent. O
Externí odkaz:
https://doaj.org/article/89015bc5e04f4dadbfb8639bce737f26
Autor:
David M Bland, B Joseph Hinnebusch
Publikováno v:
PLoS Neglected Tropical Diseases, Vol 10, Iss 2, p e0004413 (2016)
BACKGROUND:The cat flea, Ctenocephalides felis, is prevalent worldwide, will parasitize animal reservoirs of plague, and is associated with human habitations in known plague foci. Despite its pervasiveness, limited information is available about the
Externí odkaz:
https://doaj.org/article/cc97591ada6142cbbad598e394f44144
Autor:
Christopher F. Bosio, David M. Bland, Jeanette Calarco, Adélaïde Miarinjara, B. Joseph Hinnebusch
Publikováno v:
PLoS Pathogens
PLoS Pathogens, Vol 17, Iss 10, p e1009995 (2021)
PLoS Pathogens, Vol 17, Iss 10, p e1009995 (2021)
Yersinia murine toxin (Ymt) is a phospholipase D encoded on a plasmid acquired by Yersinia pestis after its recent divergence from a Yersinia pseudotuberculosis progenitor. Despite its name, Ymt is not required for virulence but acts to enhance bacte
Autor:
Shaun C Earl, Miles T Rogers, Jennifer Keen, David M Bland, Andrew S Houppert, Caitlynn Miller, Ian Temple, Deborah M Anderson, Melanie M Marketon
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0133318 (2015)
Yersinia pestis, the causative agent of bubonic and pneumonic plague, is typically a zoonotic vector-borne disease of wild rodents. Bacterial biofilm formation in the proventriculus of the flea contributes to chronic infection of fleas and facilitate
Externí odkaz:
https://doaj.org/article/265c73047c774585873031a6f232a1cd
Autor:
Forrest H. Hoyt, Dan Long, Kishore Kanakabandi, José M. C. Ribeiro, Greg Saturday, B. Joseph Hinnebusch, Rebecca Rosenke, Kimmo Virtaneva, Craig Martens, Daniel Bruno, David M. Bland
Publikováno v:
PLoS Neglected Tropical Diseases
PLoS Neglected Tropical Diseases, Vol 14, Iss 9, p e0008688 (2020)
PLoS Neglected Tropical Diseases, Vol 14, Iss 9, p e0008688 (2020)
Yersinia pestis, the causative agent of plague, is a highly lethal pathogen transmitted by the bite of infected fleas. Once ingested by a flea, Y. pestis establish a replicative niche in the gut and produce a biofilm that promotes foregut colonizatio