Zobrazeno 1 - 8
of 8
pro vyhledávání: '"David Eisenbarth"'
Publikováno v:
STAR Protocols, Vol 5, Iss 4, Pp 103354- (2024)
Summary: Evaluating the synergy of drug combinations is crucial in advancing treatment regimens. Here, we present a protocol to establish primary cells and organoids from murine tumors and calculate drug synergy. We describe all necessary cell cultur
Externí odkaz:
https://doaj.org/article/a3622ad9c92a4234a592a0cf2d5808ff
Autor:
Andreas Birbach, David Eisenbarth, Nicolas Kozakowski, Eva Ladenhauf, Marc Schmidt-Supprian, Johannes A. Schmid
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 13, Iss 8, Pp 692-703 (2011)
In prostate cancers, epidemiological data suggest a link between prostate inflammation and subsequent cancer development, but proof for this concept in a tumor model is lacking. A constitutively active version of IκB kinase 2 (IKK2), which is activa
Externí odkaz:
https://doaj.org/article/35e832bef0be4ebea027601fab2b3639
Data from YAP and AP-1 Cooperate to Initiate Pancreatic Cancer Development from Ductal Cells in Mice
The development of pancreatic cancer is heavily dependent upon the aberrant activation of KRAS signaling. Among the downstream targets of KRAS, the effectors of the Hippo pathway YAP and TAZ (YAP/TAZ) are crucial during cancer initiation and progress
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::319cb0441c78f218a295590e4022b922
https://doi.org/10.1158/0008-5472.c.6512139
https://doi.org/10.1158/0008-5472.c.6512139
Supplementary Table S1-4
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9828428de537128ab94edaf08d70ac6b
https://doi.org/10.1158/0008-5472.22425780.v1
https://doi.org/10.1158/0008-5472.22425780.v1
Autor:
David Eisenbarth, Y. Alan Wang
Publikováno v:
Journal of Cancer Metastasis and Treatment.
Glioblastoma (GBM) is one of the most immunosuppressive and heterogeneous tumors with limited treatment options. Most studies relied on treatment-experienced patient samples to elucidate the origins of tumor heterogeneity, introducing bias into the a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fbe5e01abbbdaf3cd2ad3e421299b1cf
https://doi.org/10.20517/2394-4722.2023.09
https://doi.org/10.20517/2394-4722.2023.09
Publikováno v:
Cancer Research. 80:4768-4779
The development of pancreatic cancer is heavily dependent upon the aberrant activation of KRAS signaling. Among the downstream targets of KRAS, the effectors of the Hippo pathway YAP and TAZ (YAP/TAZ) are crucial during cancer initiation and progress
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b25ab77d392374bdbdcd7de3e5a59ec
https://doi.org/10.1158/0008-5472.can-20-0907
https://doi.org/10.1158/0008-5472.can-20-0907
Autor:
Johannes A. Schmid, Nicolas Kozakowski, David Eisenbarth, Eva Ladenhauf, Andreas Birbach, Marc Schmidt-Supprian
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 13, Iss 8, Pp 692-703 (2011)
Neoplasia
Neoplasia
In prostate cancers, epidemiological data suggest a link between prostate inflammation and subsequent cancer development, but proof for this concept in a tumor model is lacking. A constitutively active version of IκB kinase 2 (IKK2), which is activa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f54e320286823eb9f0dfdf3e730eef37
https://pubmed.ncbi.nlm.nih.gov/21847361
https://pubmed.ncbi.nlm.nih.gov/21847361