Zobrazeno 1 - 10
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pro vyhledávání: '"Danila Ivanov"'
Objective: In vascular tissue, T-cadherin (T-cad) levels correlate with the progression of atherosclerosis, restenosis and tumour neovascularization. This study investigates whether T-cad influences proliferation of vascular cells. Methods and Result
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::480b5db7b098ad07e5ebe441f89a0690
http://doc.rero.ch/record/293797/files/64-1-132.pdf
http://doc.rero.ch/record/293797/files/64-1-132.pdf
Autor:
Manjunath B. Joshi, Emmanouil Kyriakakis, Paul Erne, Maria Philippova, Danila Ivanov, Therese J. Resink
Publikováno v:
Biochemical Journal. 416:271-280
T-cad (T-cadherin), a glycosylphosphatidylinositol-anchored cadherin superfamily member, is expressed widely in the brain and cardiovascular system, and absent, decreased, or even increased, in cancers. Mechanisms controlling T-cad expression are poo
Publikováno v:
Histochemistry and Cell Biology. 120:353-360
Both histological and in vitro studies indicate a relationship between T-cadherin levels and acquisition of a modulated, migratory phenotype by vascular cells. This study further examines a role for T-cadherin in relation to cell migration and adhesi
Publikováno v:
Biochemistry (Moscow). 66:1174-1186
Cadherins are a family of membrane receptors that mediate calcium-dependent homophilic cell-cell adhesion. Cadherins play a key role in the regulation of organ and tissue development during embryogenesis. In adult organisms, these proteins are respon
Autor:
Manjunath B. Joshi, Thérèse J. Resink, Paul Erne, Danila Ivanov, Maria Philippova, Roy Allenspach
Publikováno v:
The FASEB Journal. 19:1737-1739
In vascular tissue, T-cadherin (T-cad) is up-regulated in vivo under disease conditions associated with oxidative stress and concomitant cell migration, proliferation and apoptosis/survival. Using cultures of human umbilical vein endothelial cells (H
Autor:
Katharina Rupp, Manjunath B. Joshi, Paul Erne, Danila Ivanov, Therese J. Resink, Valery N. Bochkov, Maria Philippova, Emmanouil Kyriakakis, Taras Afonyushkin
Publikováno v:
Molecular and cellular biology. 28(12)
There is scant knowledge regarding how cell surface lipid-anchored T-cadherin (T-cad) transmits signals through the plasma membrane to its intracellular targets. This study aimed to identify membrane proteins colocalizing with atypical glycosylphosph
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 21(12)
Glycosylphosphatidylinositol-anchored T-cadherin (T-cad) influences several parameters of angiogenesis including endothelial cell (EC) differentiation, migration, proliferation, and survival. This presupposes signal transduction networking via mediat
Autor:
Alexander Shevelev, Danila Ivanov, T. Storozhevykh, Paul Erne, E. Talovskaya, Kseniya A. Rubina, Dmitry Stambolsky, Thérèse J. Resink, Ye. V. Parfyonova, Vsevolod A. Tkachuk, V. Pinelis, V. Cherenkov
Cadherins are a superfamily of adhesion molecules that mediate Ca2+-dependent cell–cell adhesion. T-cadherin (T-cad), a unique glycosylphosphatidylinositol-anchored member of the cadherin superfamily, was initially identified by immunoblotting of v
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b595c90bd9ff05b618d1a6b7d93d8de3
https://europepmc.org/articles/PMC1282457/
https://europepmc.org/articles/PMC1282457/
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 19(6)
T-cadherin (T-cad) is an atypical GPI-anchored member of the cadherin superfamily. Ligation of T-cad receptors on endothelial cells prevents cell spreading, promotes elongation and polarization, decreases adhesion to the matrix, and facilitates migra
Autor:
Valery N. Bochkov, Paul Erne, Julia Antropova, Thérèse J. Resink, Yelena V. Parfyonova, Vsevolod A. Tkachuk, Olga Iljinskaya, Danila Ivanov, Pavel Bashtrikov, Elena Kudrjashova, Eduard Tararak, Maria Philippova
Publikováno v:
Scopus-Elsevier
Phenotypic modulation, migration and proliferation of vascular smooth muscle cells (SMCs) are major events in restenosis after percutaneous transluminal angioplasty. Surface cell adhesion molecules, essential to morphogenesis and maintenance of adult