Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Daniela Seidinger"'
Autor:
Thomas Gremmel, Thomas Perkmann, Christoph W Kopp, Daniela Seidinger, Beate Eichelberger, Renate Koppensteiner, Sabine Steiner, Simon Panzer
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0122586 (2015)
Data linking in vivo platelet activation with inflammation and cardiovascular risk factors are scarce. Moreover, the interrelation between endothelial dysfunction as early marker of atherosclerosis and platelet activation has not been studied, so far
Externí odkaz:
https://doaj.org/article/fa2cf6f73274447c8682d12535e97e14
Autor:
Thomas Gremmel, Christoph W Kopp, Daniela Seidinger, Renate Koppensteiner, Sabine Steiner, Simon Panzer
Publikováno v:
Swiss Medical Weekly, Vol 143, Iss 2122 (2013)
BACKGROUND: Atherosclerosis affects many patients with type 2 diabetes. Both are associated with platelet activation, but it remains unclear how diabetes contributes to, or even enhances, platelet activation in patients with atherosclerosis. We there
Externí odkaz:
https://doaj.org/article/fb68438eb170449cafe8ae661492086e
Autor:
Alexandra Hammer, Manuela Matschuck, Renate Koppensteiner, Daniela Seidinger, Oliver Schlager, Sabine Steiner, Deddo Moertl
Publikováno v:
British Journal of Nutrition. 122:698-706
As only limited evidence is available for potential benefits of n-3 PUFA supplementation in patients with peripheral arterial disease (PAD), we studied the effects of 4 g n-3 PUFA on endothelial function and inflammatory markers. Seventy patients wit
Autor:
Thomas Gremmel, Simon Panzer, Irene M. Lang, Beate Eichelberger, Joseph Pultar, Patricia P. Wadowski, Renate Koppensteiner, Daniela Seidinger, Christoph W. Kopp
Publikováno v:
Journal of Cardiovascular Translational Research
Disaggregation as the difference between maximal and final platelet aggregation by light transmission aggregometry indicates the stability of platelet aggregates. We evaluated the extent of disaggregation after platelet stimulation with adenosine dip
Autor:
Alexandra, Hammer, Deddo, Moertl, Oliver, Schlager, Manuela, Matschuck, Daniela, Seidinger, Renate, Koppensteiner, Sabine, Steiner
Publikováno v:
The British journal of nutrition. 122(6)
As only limited evidence is available for potential benefits of n-3 PUFA supplementation in patients with peripheral arterial disease (PAD), we studied the effects of 4 g n-3 PUFA on endothelial function and inflammatory markers. Seventy patients wit
Autor:
Renate Koppensteiner, Oliver Schlager, Daniela Seidinger, Sonja Zehetmayer, Bernd van der Loo
Publikováno v:
Atherosclerosis. 233:76-82
Local changes in wall shear stress (WSS) contribute to vascular wall thickening and subsequent stenosis. Restenosis after stenting is a major concern, especially in the superficial femoral artery (SFA) of patients with peripheral arterial disease (PA
Autor:
Renate Koppensteiner, Christoph W. Kopp, Simon Panzer, Daniela Seidinger, Thomas Gremmel, Sabine Steiner
Publikováno v:
Thrombosis and Haemostasis. 112:474-482
SummaryData linking the response to antiplatelet therapy with clinical outcomes after angioplasty and stenting for lower extremity artery disease (LEAD) are scarce. Moreover, associations of in vivo and thrombin-inducible platelet activation with the
Autor:
Thomas Gremmel, Sabine Steiner, Renate Koppensteiner, Simon Panzer, Daniela Seidinger, Christoph W. Kopp
Publikováno v:
European Journal of Clinical Investigation. 43:689-697
Background Abundant thrombin generation may be a major reason for subsequent thromboembolic events in patients with cardiovascular disease receiving dual antiplatelet therapy. We therefore investigated the susceptibility of thienopyridine responders
Autor:
Thomas Perkmann, Simon Panzer, Sabine Steiner, Renate Koppensteiner, Daniela Seidinger, Thomas Gremmel, Christoph W. Kopp
Publikováno v:
Journal of Atherosclerosis and Thrombosis. 20:630-645
AIMS Inflammation has been postulated to modify the platelet response to aspirin treatment, thereby causing high on-treatment residual platelet reactivity (HRPR). Both high levels of inflammatory markers and HRPR have been linked to adverse cardiovas
Autor:
Renate Koppensteiner, Christoph W. Kopp, Daniela Seidinger, Sabine Steiner, Simon Panzer, Thomas Gremmel
Publikováno v:
International Journal of Cardiology. 160:109-113
Background Patients with high on-treatment residual adenosine diphosphate-inducible platelet reactivity (HRPR) despite clopidogrel therapy are at an increased risk for adverse events after coronary stenting. A higher maintenance dose of clopidogrel m