Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Daniela SANDU"'
Autor:
Elena-Daniela SANDU
Publikováno v:
Studia Universitatis Babeş-Bolyai. Philologia, Vol 65, Iss 3 (2020)
The Jungian Archetypes and the Conflict with the Double Self in The Strange Case of Dr. Jekyll and Mr. Hyde. The current paper analyzes Robert Louis Stevenson’s novella The Strange Case of Dr. Jekyll and Mr. Hyde from a psychoanalytical perspective
Externí odkaz:
https://doaj.org/article/8dccf5873f6f436abaf7a0f0395bbf08
Autor:
Daniela Sandu, Editura Universitara
Prin finalizarea cercetarii de fata ne-am propus sa oferim cadrelor didactice instrumente de lucru in organizarea activitatilor pe echipe si mai ales modalitati de evaluare a procesului invatarii prin cooperare. Iar la sfarsitul stradaniilor lor sa a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1c2fea87c1a74cdf64d6b7913a67855b
https://doi.org/10.5682/9786062811327
https://doi.org/10.5682/9786062811327
Autor:
T. Sreenath, Ruth H. Walker, C. W. Olanow, Donald J. Weisz, Ioanna A. Armata, P. Shashidharan, Daniela Sandu, Mitchell F. Brin, U. Potla, Kevin St. P. McNaught
Publikováno v:
Human Molecular Genetics. 14:125-133
Early-onset dystonia is an autosomal dominant movement disorder associated with deletion of a glutamic acid residue in torsinA. We generated four independent lines of transgenic mice by overexpressing human DeltaE-torsinA using a neuron specific enol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa36f66fc828156b07974ee7d11d1c06
https://doi.org/10.1093/hmg/ddi012
https://doi.org/10.1093/hmg/ddi012
Publikováno v:
Neurology. 58:120-124
A mutation of the DYT1 gene, which codes for torsinA, has been identified as the cause of one form of autosomal dominantly inherited dystonia. TorsinA immunohistochemistry was used to examine a case of DYT1, and several cases of non-DYT1, dystonia. N
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2440ab73fef191dc7619106f7db2a01f
https://doi.org/10.1212/wnl.58.1.120
https://doi.org/10.1212/wnl.58.1.120
The paper presents an e-Learning platform in project management centred on the learner and based on advanced technologies, such as: ontology, competence standards, IMS Standards.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______645::734fd09631228d9b0cbafe01f7a52b35
http://ccasp.ase.ro/KM2009/38.pdf
http://ccasp.ase.ro/KM2009/38.pdf
Autor:
P, Shashidharan, Nicolae, Paris, Daniela, Sandu, Laina, Karthikeyan, Kevin St P, McNaught, Ruth H, Walker, C Warren, Olanow
Publikováno v:
Journal of neurochemistry. 88(4)
Childhood-onset dystonia is an autosomal dominant movement disorder associated with a three base pair (GAG) deletion mutation in the DYT1 gene. This gene encodes a novel ATP-binding protein called torsinA, which in the central nervous system is expre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::348f7d6b2735d524849c7bef7654de63
https://pubmed.ncbi.nlm.nih.gov/14756824
https://pubmed.ncbi.nlm.nih.gov/14756824
Publikováno v:
Advances in Behavioral Biology ISBN: 9781461352075
A mutation of the DYT1 gene, which codes for torsinA, has been identified as the cause of one form of autosomal-dominantly inherited dystonia. We used a recently-developed polyclonal antibody to examine torsinA immunohistochemistry in the brain of a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f84fd2fdec7097f82681432b4f240da4
https://doi.org/10.1007/978-1-4615-0715-4_51
https://doi.org/10.1007/978-1-4615-0715-4_51
Autor:
Patrick R. Hof, Pushpa Gujjari, C. Warren Olanow, Daniela Sandu, P. Shashidharan, Mitchell F. Brin, Ruth H. Walker
Publikováno v:
Brain research. 900(2)
A mutation of the DYT1 gene on chromosome 9q34 has recently been identified as the cause of one form of autosomal-dominantly inherited dystonia. TorsinA, the protein product of this gene, has homology with the family of heat shock proteins, and is fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d7a3be38a990a95c0349bfca7bce4a44
https://pubmed.ncbi.nlm.nih.gov/11334819
https://pubmed.ncbi.nlm.nih.gov/11334819