Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Daniela R. Macció"'
Autor:
Osvaldo L. Podhajcer, Guillermo Mazzolini, Cecilia Carbone, Diego L. Viale, Maria V. Lopez, Daniela R. Macció, Eduardo G. Cafferata
Purpose: A33 antigen is a membrane-bound protein expressed in intestinal epithelium that is overexpressed in 95% of primary and metastatic colorectal carcinomas but is absent in most epithelial tissues and tumor types. We hypothesized that A33 promot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4052b4259e453780570b9e816edc083c
https://doi.org/10.1158/1078-0432.c.6517545.v1
https://doi.org/10.1158/1078-0432.c.6517545.v1
Autor:
Osvaldo L. Podhajcer, Guillermo Mazzolini, Cecilia Carbone, Diego L. Viale, Maria V. Lopez, Daniela R. Macció, Eduardo G. Cafferata
Supplementary Data from A Novel A33 Promoter–Based Conditionally Replicative Adenovirus Suppresses Tumor Growth and Eradicates Hepatic Metastases in Human Colon Cancer Models
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::18b90c5b9d3422b184d7c58f82350ad7
https://doi.org/10.1158/1078-0432.22440177
https://doi.org/10.1158/1078-0432.22440177
Publikováno v:
Journal of Neuroimmunology. 199:63-66
This study assessed whether the in vitro effect of testosterone on the proliferative response of mononuclear cells to myelin basic protein (MBP) could be mediated by nitric oxide (NO). Testosterone but not cholesterol supplementation specifically sup
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1703885f2666410e41dd6f70185f094
https://doi.org/10.1016/j.jneuroim.2008.05.005
https://doi.org/10.1016/j.jneuroim.2008.05.005
Autor:
Juan Pablo Nicola, Ana María Masini-Repiso, German A. Roth, Laura Fozzatti, Claudia Gabriela Pellizas, Ariel Maximiliano Lucero, María Laura Vélez, Ivan D. Mascanfroni, Daniela R. Macció
Publikováno v:
The Journal of endocrinology. 192(3)
Nitric oxide (NO) is a free radical that mediates a wide array of cell functions. It is generated from l-arginine by NO-synthase (NOS). Expression of NOS isoforms has been demonstrated in thyroid cells. Previous reports indicated that NO donors induc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7eed13ba0834dd6ccccba0a73434c5bf
https://pubmed.ncbi.nlm.nih.gov/17332530
https://pubmed.ncbi.nlm.nih.gov/17332530
Publikováno v:
FABICIB; Vol. 7 (2003): FABICIB; 25-36
FABICIB; Vol 7 (2003): FABICIB; 25-36
Biblioteca Virtual (UNL)
Universidad Nacional del Litoral
instacron:UNL
FABICIB; Vol 7 (2003): FABICIB; 25-36
Biblioteca Virtual (UNL)
Universidad Nacional del Litoral
instacron:UNL
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b497d692884c14a500a1bd68f6431b3e
https://bibliotecavirtual.unl.edu.ar/publicaciones/index.php/FABICIB/article/view/715
https://bibliotecavirtual.unl.edu.ar/publicaciones/index.php/FABICIB/article/view/715
Publikováno v:
Autoimmunity. 37(1)
Experimental autoimmune encephalomyelitis (EAE) is an inflammatory disease of the CNS mediated by autoreactive T lymphocytes directed against myelin antigens. Since neuroendocrine-immune dysfunction appears to contribute to the pathogenesis of autoim
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c6b9c1ed7c509b26453f2dd05716d2d
https://pubmed.ncbi.nlm.nih.gov/15115307
https://pubmed.ncbi.nlm.nih.gov/15115307
Publikováno v:
Neuro endocrinology letters. 25(3)
Experimental autoimmune encephalomyelitis (EAE) was induced to investigate the levels of circulating total testosterone (TT) and the possible association of corticosterone with the steroid-producing capacity of the testes.We determined gonad weights,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b7f3ce3b5d8f1bb520d915c250a13c9d
https://pubmed.ncbi.nlm.nih.gov/15349085
https://pubmed.ncbi.nlm.nih.gov/15349085
Autor:
Monte Stevens
p53: Structure, Functions and Role in Disease discusses the role of p53 dysregulation in different hematologic neoplasms defined by the current WHO classification. The prevalence of p53 aberration, mechanisms of p53 inactivation, regulation of p53 si