Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Daniela Pereira Antelo"'
Publikováno v:
Anais Brasileiros de Dermatologia, Vol 83, Iss 6, Pp 572-574 (2008)
Na patogênese do vitiligo tem-se enfatizado o papel das células T citotóxicas. Identificadas pelo antígeno linfocitário cutâneo (CLA), essas células já foram descritas no sangue de pacientes com outras dermatoses e podem ser depletadas pela f
Externí odkaz:
https://doaj.org/article/425cd90fc7ef497499ce7a54716f2044
Publikováno v:
Photodermatology, Photoimmunology & Photomedicine. 27:40-44
Background: Vitiligo is a frequently acquired, hereditary disease, characterized by achromic macules due to the absence of melanocytes. In contrast with earlier studies, in which the main pathogenic role was attributed to anti-melanocyte antibodies,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6e0fd0a1d3e1c276c48ccf7af60c415e
https://doi.org/10.1111/j.1600-0781.2010.00563.x
https://doi.org/10.1111/j.1600-0781.2010.00563.x
Immunohistochemical Findings in Active Vitiligo Including Depigmenting Lesions and Non-Lesional Skin
Autor:
Ibrahim Galadari, Rogerio E. Farias, Flavia M. N. P. Aslanian, Daniela Pereira Antelo, Pranab K. Das, Tullia Cuzzi, Absalom Lima Filgueira, Rosangela Aparecida M. Noé
Publikováno v:
The Open Dermatology Journal. 2:105-110
Background: The evidence that vitiligo is an autoimmune disease is supported by its association with autoimmune conditions, the presence of activated cytotoxic T lymphocytes in the lesions and melanocyte-specific circulating auto-antibodies. Some stu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d7a38c6fc125f97cd48f81b9b0905b39
https://doi.org/10.2174/1874372200802010105
https://doi.org/10.2174/1874372200802010105
Autor:
Elisa Estrella, Vania Cesario de Souza, Daniela Pereira Antelo, Luna Azulay-Abulafia, Maria Auxiliadora Jeunon de Sousa
Publikováno v:
Anais Brasileiros de Dermatologia. 78:611-613
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e5ed319466efc751a23182ef17084022
https://doi.org/10.1590/s0365-05962003000500011
https://doi.org/10.1590/s0365-05962003000500011