Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Daniela Díaz-Catalán"'
Autor:
Laura Boswell, Arturo Vega-Beyhart, Miquel Blasco, Luis F. Quintana, Gabriela Rodríguez, Daniela Díaz-Catalán, Carme Vilardell, María Claro, Mireia Mora, Antonio J. Amor, Gregori Casals, Felicia A. Hanzu
Publikováno v:
Frontiers in Endocrinology, Vol 15 (2024)
ObjectiveWe compared hair cortisol (HC) with classic tests of the hypothalamic–pituitary–adrenal (HPA) axis in chronic kidney disease (CKD) and assessed its association with kidney and cardiometabolic status.Design and methodsA cross-sectional st
Externí odkaz:
https://doaj.org/article/6f53a88b90484c888c5b0c5374728f45
Autor:
Sara de Pablo, Júlia Rodríguez-Comas, Daniela Díaz-Catalán, Gema Alcarraz-Vizán, Carlos Castaño, Juan Moreno-Vedia, Joel Montane, Marcelina Parrizas, Joan-Marc Servitja, Anna Novials
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Abstract Amyloid deposits in pancreatic islets, mainly formed by human islet amyloid polypeptide (hIAPP) aggregation, have been associated with loss of β-cell mass and function, and are a pathological hallmark of type 2 diabetes (T2D). Treatment wit
Externí odkaz:
https://doaj.org/article/0369fa00d1f7444587242a36dc3bc794
Autor:
Arturo Vega-Beyhart, Javier Laguna-Moreno, Daniela Díaz-Catalán, Laura Boswell, Mireia Mora, Irene Halperin, Gregori Casals, Felicia A. Hanzu
Publikováno v:
Frontiers in Endocrinology, Vol 13 (2022)
IntroductionTwenty-four-hour urinary free cortisol (24h-UFC) is the most used test for follow-up decision-making in patients with Cushing syndrome (CS) under medical treatment. However, 24h-UFC determinations by immunoassays (IA) are commonly overest
Externí odkaz:
https://doaj.org/article/b878dd68d03145e5afd31d8afefadef2
Autor:
Daniela Díaz-Catalán, Gema Alcarraz-Vizán, Carlos Castaño, Sara de Pablo, Júlia Rodríguez-Comas, Antonio Fernández-Pérez, Mario Vallejo, Sara Ramírez, Marc Claret, Marcelina Parrizas, Anna Novials, Joan-Marc Servitja
Publikováno v:
Molecular Metabolism, Vol 53, Iss , Pp 101251- (2021)
Objective: Pancreatic β-cell dysfunction is a central feature in the pathogenesis of type 2 diabetes (T2D). Accumulating evidence indicates that β-site APP-cleaving enzyme 2 (BACE2) inhibition exerts a beneficial effect on β-cells in different mod
Externí odkaz:
https://doaj.org/article/e1d707b7867642fd9e5b8a8015043964
Autor:
Júlia Rodríguez-Comas, Juan Moreno-Vedia, Mercè Obach, Carlos Castaño, Sara de Pablo, Gema Alcarraz-Vizán, Daniela Díaz-Catalán, Anna Mestre, Raquel Horrillo, Montserrat Costa, Anna Novials, Joan-Marc Servitja
Publikováno v:
Molecular Metabolism, Vol 37, Iss , Pp 100984- (2020)
Objective: Pancreatic β-cell failure is central to the development and progression of type 2 diabetes (T2D). The aggregation of human islet amyloid polypeptide (hIAPP) has been associated with pancreatic islet inflammation and dysfunction in T2D. Al
Externí odkaz:
https://doaj.org/article/a5c1a13a452440598ccd2dc0ac96e99b
Autor:
Joan-Marc Servitja, Marcelina Párrizas, Carlos Castaño, Gema Alcarraz-Vizán, Daniela Díaz-Catalán, Joel Montane, Júlia Rodríguez-Comas, Anna Novials, Juan Moreno-Vedia, Sara de Pablo
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Scientific Reports
Scientific Reports
Amyloid deposits in pancreatic islets, mainly formed by human islet amyloid polypeptide (hIAPP) aggregation, have been associated with loss of β-cell mass and function, and are a pathological hallmark of type 2 diabetes (T2D). Treatment with chapero
Autor:
Carlos Castaño, Gema Alcarraz-Vizán, Marcelina Párrizas, Sara de Pablo, Júlia Rodríguez-Comas, Joan-Marc Servitja, Mario Vallejo, Antonio Fernández-Pérez, Sara Ramírez, Daniela Díaz-Catalán, Anna Novials, Marc Claret
Publikováno v:
Molecular Metabolism
Digital.CSIC. Repositorio Institucional del CSIC
instname
Molecular Metabolism, Vol 53, Iss, Pp 101251-(2021)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Molecular Metabolism, Vol 53, Iss, Pp 101251-(2021)
© 2021 The Author(s).
[Objective]: Pancreatic β-cell dysfunction is a central feature in the pathogenesis of type 2 diabetes (T2D). Accumulating evidence indicates that β-site APP-cleaving enzyme 2 (BACE2) inhibition exerts a beneficial effec
[Objective]: Pancreatic β-cell dysfunction is a central feature in the pathogenesis of type 2 diabetes (T2D). Accumulating evidence indicates that β-site APP-cleaving enzyme 2 (BACE2) inhibition exerts a beneficial effec
Autor:
Mercè Obach, Gema Alcarraz-Vizán, Júlia Rodríguez-Comas, Juan Moreno-Vedia, Anna Novials, Raquel Horrillo, Joan-Marc Servitja, Montserrat Costa, Carlos Castaño, Daniela Díaz-Catalán, Sara de Pablo, Anna Mestre
Publikováno v:
Molecular Metabolism, Vol 37, Iss, Pp 100984-(2020)
Molecular Metabolism
Molecular Metabolism
Objective Pancreatic β-cell failure is central to the development and progression of type 2 diabetes (T2D). The aggregation of human islet amyloid polypeptide (hIAPP) has been associated with pancreatic islet inflammation and dysfunction in T2D. Alp