Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Daniela, Rosenhauer"'
Autor:
Anna-Carolina Friedrich, Peter Linz, Armin M. Nagel, Daniela Rosenhauer, Stephan Horn, Mario Schiffer, Michael Uder, Christoph Kopp, Anke Dahlmann
Publikováno v:
Kidney & Blood Pressure Research (2021)
Background: The relationship between Na+ balance and cardiovascular disease (CVD) in hemodialysis (HD) patients is not yet fully understood. We hypothesized that HD patients co-diagnosed with CVD show increased tissue Na+ accumulation compared to HD
Externí odkaz:
https://doaj.org/article/a5bb3dfe01b842dd8cc5f6fa9b6ed6a8
Autor:
Mario Schiffer, Manfred Rauh, Friedrich C. Luft, Armin M. Nagel, Christoph W. Kopp, Stephan Horn, Peter Linz, Isabelle Zucker, Daniela Rosenhauer, Jonathan Jantsch, Dominik N. Müller, Michael Uder, Anke Dahlmann, Thomas Dienemann, Viktor Haag, Patrick Neubert
Publikováno v:
Kidney International Reports
Introduction Chronic kidney disease (CKD) engenders salt-sensitive hypertension. Whether or not tissue Na+ accumulation is increased in CKD patients remains uncertain. How tissue Na+ is affected after renal transplantation has not been assessed. Meth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61cbafd07c90f380134feecc0c1a53b3
https://doi.org/10.1016/j.ekir.2021.06.022
https://doi.org/10.1016/j.ekir.2021.06.022
Autor:
Friedrich C. Luft, Peter Wabel, Peter Linz, Stephan Horn, Daniela Rosenhauer, Michael Uder, Christoph W. Kopp, Armin M. Nagel, Carolin Maier, Anke Dahlmann, Matthias Hammon, Jens Titze
Publikováno v:
Kidney International. 93:1191-1197
Long-term elevated blood sugar levels result in tissue matrix compositional changes in patients with diabetes mellitus type 2 (T2DM). We hypothesized that hemodialysis patients with T2DM might accumulate more tissue sodium than control hemodialysis p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dbb83b0571bb40bc377c509f33660f66
https://doi.org/10.1016/j.kint.2017.11.021
https://doi.org/10.1016/j.kint.2017.11.021
Autor:
Jens Titze, Peter Linz, Anke Dahlmann, Matthias Hammon, Hannes Seuss, Michael Uder, Susan Grossmann, Rolf Janka, Rebecca Hammon, Alexander Cavallaro, Friedrich C. Luft, Daniela Rosenhauer
Publikováno v:
Academic Radiology. 24:1086-1093
Rationale and Objectives Sodium and proton magnetic resonance imaging (23Na/1H-MRI) have shown that muscle and skin can store Na+ without water. In chronic renal failure and in heart failure, Na+ mobilization occurs, but is variable depending on age,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f02973ab03e5f2625319341893533d05
https://doi.org/10.1016/j.acra.2017.03.012
https://doi.org/10.1016/j.acra.2017.03.012
Autor:
Daniela Rosenhauer, Matthias Hammon, Kai-Uwe Eckardt, Alexander Cavallaro, Georg Schett, Christian Beyer, Jonathan Jantsch, Michael Uder, Dominik N. Müller, Anke Dahlmann, Jörg H W Distler, Friedrich C. Luft, Patrick Neubert, Jens Titze, Peter Linz, Christoph W. Kopp
Publikováno v:
Rheumatology. 56:556-560
Objective. Skin fibrosis is the predominant feature of SSc and arises from excessive extracellular matrix deposition. Glycosaminoglycans are macromolecules of the extracellular matrix, which facilitate Na(+) accumulation in the skin. We used (23)Na-M
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9bb944857fc1cb5042f1091c6adcc7e0
https://doi.org/10.1093/rheumatology/kew371
https://doi.org/10.1093/rheumatology/kew371
Autor:
Christoph, Kopp, Peter, Linz, Carolin, Maier, Peter, Wabel, Matthias, Hammon, Armin M, Nagel, Daniela, Rosenhauer, Stephan, Horn, Michael, Uder, Friedrich C, Luft, Jens, Titze, Anke, Dahlmann
Publikováno v:
Kidney international. 93(5)
Long-term elevated blood sugar levels result in tissue matrix compositional changes in patients with diabetes mellitus type 2 (T2DM). We hypothesized that hemodialysis patients with T2DM might accumulate more tissue sodium than control hemodialysis p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b6eaf0f49ed7a1c6cb38d2c1191776c7
https://pubmed.ncbi.nlm.nih.gov/29455909
https://pubmed.ncbi.nlm.nih.gov/29455909