Zobrazeno 1 - 10
of 37
pro vyhledávání: '"Daniel J. Beard"'
Publikováno v:
Frontiers in Stroke, Vol 2 (2023)
Externí odkaz:
https://doaj.org/article/5d37206f0c66482a93282a15bae0644a
Autor:
Daniel Omileke, Sara Azarpeykan, Steven W. Bothwell, Debbie Pepperall, Daniel J. Beard, Kirsten Coupland, Adjanie Patabendige, Neil J. Spratt
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-9 (2021)
Abstract Reperfusion therapies re-establish blood flow after arterial occlusion and improve outcome for ischaemic stroke patients. Intracranial pressure (ICP) elevation occurs 18–24 h after experimental stroke. This elevation is prevented by short-
Externí odkaz:
https://doaj.org/article/92a4ab37e1844ea2bc81fdc3d482f947
Autor:
Sonia Sanchez-Bezanilla, Daniel J. Beard, Rebecca J. Hood, N. David Åberg, Patricia Crock, Frederick R. Walker, Michael Nilsson, Jörgen Isgaard, Lin Kooi Ong
Publikováno v:
Neural Plasticity, Vol 2022 (2022)
Aims. We have shown that growth hormone (GH) treatment poststroke increases neuroplasticity in peri-infarct areas and the hippocampus, improving motor and cognitive outcomes. We aimed to explore the mechanisms of GH treatment by investigating how GH
Externí odkaz:
https://doaj.org/article/9e88a0a021a4457c9225111fcaedfb0b
Autor:
Daniel Omileke, Debbie Pepperall, Steven W. Bothwell, Nikolce Mackovski, Sara Azarpeykan, Daniel J. Beard, Kirsten Coupland, Adjanie Patabendige, Neil J. Spratt
Publikováno v:
Frontiers in Neurology, Vol 12 (2021)
There is a transient increase in intracranial pressure (ICP) 18–24 h after ischaemic stroke in rats, which is prevented by short-duration hypothermia using rapid cooling methods. Clinical trials of long-duration hypothermia have been limited by fea
Externí odkaz:
https://doaj.org/article/143733157fac49edb8a0231418e87c47
Publikováno v:
Frontiers in Physiology, Vol 12 (2021)
Externí odkaz:
https://doaj.org/article/58af2c433475464e9c04c38d41f6b3be
Autor:
Daniel Omileke, Steven W. Bothwell, Debbie Pepperall, Daniel J. Beard, Kirsten Coupland, Adjanie Patabendige, Neil J. Spratt
Publikováno v:
Brain Sciences, Vol 11, Iss 12, p 1589 (2021)
Background: Elevated intracranial pressure (ICP) occurs 18–24 h after ischaemic stroke and is implicated as a potential cause of early neurological deterioration. Increased resistance to cerebrospinal fluid (CSF) outflow after ischaemic stroke is a
Externí odkaz:
https://doaj.org/article/6d250e56b68c4c3882450a19dde75d1d
Autor:
Bryan A. Adriaanse, Sinead Brady, Minghui Wang, Daniel J. Beard, Jonathan I. Spencer, Jonathan Pansieri, Brad A. Sutherland, Margaret M. Esiri, Alastair M. Buchan, Zameel Cader, Bin Zhang, Gabriele C. DeLuca
Publikováno v:
Neuropathology and Applied Neurobiology. 49
Autor:
Rebecca J. Hood, Sonia Sanchez-Bezanilla, Daniel J. Beard, Ruslan Rust, Renée J. Turner, Shannon M. Stuckey, Lyndsey E. Collins-Praino, Frederick R. Walker, Michael Nilsson, Lin Kooi Ong
We have previously demonstrated that a cortical stroke causes persistent impairment of hippocampal-dependent cognitive tasks concomitant with secondary neurodegenerative processes such as amyloid-β accumulation in the hippocampus, a region remote fr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e48e8f9e9633bb0d1ec9f4d4c9fc53dd
https://doi.org/10.1101/2023.04.24.538047
https://doi.org/10.1101/2023.04.24.538047
Publikováno v:
Frontiers in Stroke. 2
Intracranial pressure (ICP) elevation post-stroke has long been thought of as a cause of secondary deterioration after large, malignant infarction, and dramatic ICP elevation is frequently a pre-terminal event. However, there is an increasing body of
Autor:
Steven W. Bothwell, Daniel J. Beard, Kirsten Coupland, Daniel Omileke, Neil J. Spratt, Adjanie Patabendige, Debbie Pepperall
Publikováno v:
Brain Sciences; Volume 11; Issue 12; Pages: 1589
Brain Sciences, Vol 11, Iss 1589, p 1589 (2021)
Brain Sciences
BRAIN SCIENCES
Brain Sciences, Vol 11, Iss 1589, p 1589 (2021)
Brain Sciences
BRAIN SCIENCES
Background: Elevated intracranial pressure (ICP) occurs 18–24 h after ischaemic stroke and is implicated as a potential cause of early neurological deterioration. Increased resistance to cerebrospinal fluid (CSF) outflow after ischaemic stroke is a