Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Dallas Turley"'
Autor:
Yufen Chen, Timothy J. Carroll, Kelly D. Flemming, Chandra Y. Gerrard, Julián Carrión-Penagos, John Huston, Yunhong Shu, Atif Zafar, Janne Koskimäki, Sean P. Polster, Issam A. Awad, Ying Cao, Reed Selwyn, Marc C. Mabray, Robert Shenkar, James I. Koenig, Todd B. Parrish, Xiaohong Joe Zhou, Romuald Girard, Agnieszka Stadnik, Alexey Dimov, Nicholas Hobson, Dallas Turley
Publikováno v:
J Magn Reson Imaging
Background Quantitative susceptibility mapping (QSM) and dynamic contrast-enhanced quantitative permeability (DCEQP) on magnetic resonance (MR) have been shown to correlate with neurovascular disease progression as markers of vascular leakage and hem
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5353ccb10f07fa507235d01b7f2f2921
https://pubmed.ncbi.nlm.nih.gov/31515878
https://pubmed.ncbi.nlm.nih.gov/31515878
Autor:
Agnieszka Stadnik, Leslie Morrison, Daniel F. Hanley, Reed Selwyn, John Huston, Nicholas Hobson, Dallas Turley, Kelly D. Flemming, Janne Koskimäki, Ying Cao, Sean P. Polster, Romuald Girard, Timothy J. Carroll, Issam A. Awad, Robert Shenkar
Publikováno v:
Stroke. 50
Background: Cerebral cavernous malformation is a genetic neurovascular disorder causing aberrant angiogenesis and lesion development within capillary beds. Lesions exhibit impaired endothelial barrier function, causing blood-brain barrier leakage and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::51d1f1d100f2b12af036b05161cf328f
https://doi.org/10.1161/str.50.suppl_1.tp582
https://doi.org/10.1161/str.50.suppl_1.tp582
Autor:
Dror Harats, Dallas Turley, Ararat Chakhoyan, Nicholas Butowski, Caleb Tan, Yael C Cohen, Andrew Brenner, Jacob Schlossman, Timothy F. Cloughesy, Tamar Rachmilewitz Minei, Jodi Goldman, Benjamin M. Ellingson, Joseph Tsung, Jingwen Yao, Patrick Y. Wen, Catalina Raymond
Publikováno v:
Journal of Clinical Oncology. 37:2018-2018
2018 Background: VB-111 is a non-replicating adenovirus carrying a pro-apoptotic transgene for TNFR1/Fas under the control of a modified murine promoter to pre-proendothelin 1. The transgene is expressed only in angiogenic endothelial cells, and ther
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d127a9f79cf137c8ff504c061f6f89a5
https://doi.org/10.1200/jco.2019.37.15_suppl.2018
https://doi.org/10.1200/jco.2019.37.15_suppl.2018