Zobrazeno 1 - 10
of 68
pro vyhledávání: '"Dale M Seth"'
Publikováno v:
PLoS ONE, Vol 8, Iss 3, p e58339 (2013)
The binding of renin or prorenin to the (pro)renin receptor (PRR) promotes angiotensin (Ang) II formation and mediates Ang II-independent signaling pathways. In the central nervous system (CNS), Ang II regulates blood pressure via inducing oxidative
Externí odkaz:
https://doaj.org/article/cdd4a632df794b98a8c6fbf836ab632c
Publikováno v:
Physiological Reports, Vol 7, Iss 24, Pp n/a-n/a (2019)
Abstract Chymase released from mast cells produces pro‐fibrotic, inflammatory, and vasoconstrictor agents. Studies were performed to test the hypothesis that chronic chymase inhibition provides a renal protective effect in type 2 diabetes. Diabetic
Externí odkaz:
https://doaj.org/article/2acfb1a8544f40c18033dd3b620fe79d
Publikováno v:
International Journal of Hypertension, Vol 2012 (2012)
It is well known that the brain renin-angiotensin (RAS) system plays an essential role in the development of hypertension, mainly through the modulation of autonomic activities and vasopressin release. However, how the brain synthesizes angiotensin (
Externí odkaz:
https://doaj.org/article/2a0c3d8bcde246a4b56f60eae3586609
Publikováno v:
Physiological Reports, Vol 7, Iss 24, Pp n/a-n/a (2019)
Physiological Reports
Physiological Reports
Chymase released from mast cells produces pro‐fibrotic, inflammatory, and vasoconstrictor agents. Studies were performed to test the hypothesis that chronic chymase inhibition provides a renal protective effect in type 2 diabetes. Diabetic (db/db)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2c8ee0f96fbd4958b934e44722e9cb1
https://doi.org/10.14814/phy2.14302
https://doi.org/10.14814/phy2.14302
Autor:
Kenneth D. Mitchell, Ryousuke Satou, Weijian Shao, Minolfa C. Prieto, L. Gabriel Navar, Carla B. Rosales, Dale M. Seth, Kayoko Miyata, Akemi Katsurada
Publikováno v:
American Journal of Physiology-Renal Physiology. 311:F278-F290
In angiotensin II (ANG II)-dependent hypertension, there is an angiotensin type 1 receptor-dependent amplification mechanism enhancing intrarenal angiotensinogen (AGT) formation and secretion in the tubular fluid. To evaluate the role of increased ar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::49b79ca2b72cc391dae05d2b3e74f6c1
https://doi.org/10.1152/ajprenal.00419.2015
https://doi.org/10.1152/ajprenal.00419.2015
Autor:
Cristobal Ibaceta-Gonzalez, Liu Liu, Dale M. Seth, Camille R. T. Bourgeois, Nicolas Salinas-Parra, Minolfa C. Prieto, Venkateswara R Gogulamudi, Lucienne S. Lara, Alexis A. Gonzalez
Publikováno v:
American Journal of Physiology-Renal Physiology. 309:F880-F888
In contrast to the negative feedback of angiotensin II (ANG II) on juxtaglomerular renin, ANG II stimulates renin in the principal cells of the collecting duct (CD) in rats and mice via ANG II type 1 (AT1R) receptor, independently of blood pressure.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cc4bc66b58ef944cfe8dd19da64ab6a6
https://doi.org/10.1152/ajprenal.00155.2015
https://doi.org/10.1152/ajprenal.00155.2015
Publikováno v:
The American Journal of the Medical Sciences. 348:416-422
Background Evidence indicates that chronic angiotensin II (AngII) infusion increases (pro)renin receptor ((P)RR) expression in renal inner medullary collecting duct (IMCD) cells. Recently, it has been shown that renal (P)RR expression is augmented du
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::327e79a484d255d88380b85b4401f667
https://doi.org/10.1097/maj.0000000000000335
https://doi.org/10.1097/maj.0000000000000335
Autor:
Alicia A. McDonough, Anne Riquier-Brison, Mien T. X. Nguyen, Kenneth E. Bernstein, Nicolas Picard, Sebastien Fuchs, Tea Janjoulia, Dominique Eladari, L. Gabriel Navar, Jorge F. Giani, Eric Delpire, Sebastian Bachmann, Romer A. Gonzalez-Villalobos, Dale M. Seth, János Peti-Peterdi, Nicholas K. Fletcher
Publikováno v:
Journal of Clinical Investigation. 123:2011-2023
Activation of the intrarenal renin-angiotensin system (RAS) can elicit hypertension independently from the systemic RAS. However, the precise mechanisms by which intrarenal Ang II increases blood pressure have never been identified. To this end, we s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::511c765cf70056bde55018216666a3d1
https://doi.org/10.1172/jci65460
https://doi.org/10.1172/jci65460
Autor:
Fernanda Barrinha Fernandes, Andreia Cristina Febba Gomes, Dale M. Seth, Zaira Palomino Jara, Minolfa C. Prieto, L. Gabriel Navar, Dulce Elena Casarini
Publikováno v:
New Aspects of the Renin Angiotensin System in Cardiovascular and Renal Diseases
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0357e8b743d6aefb115dabf67806f567
https://doi.org/10.2174/9781681083131116010019
https://doi.org/10.2174/9781681083131116010019
Publikováno v:
International Journal of Hypertension
International Journal of Hypertension, Vol 2012 (2012)
International Journal of Hypertension, Vol 2012 (2012)
It is well known that the brain renin-angiotensin (RAS) system plays an essential role in the development of hypertension, mainly through the modulation of autonomic activities and vasopressin release. However, how the brain synthesizes angiotensin (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::703d79b124db68ad193e72d0e0376f64
https://doi.org/10.1155/2012/290635
https://doi.org/10.1155/2012/290635