Výsledky vyhledávání - "Daisuke Nambara"

Akademický článek

CiAPEX2 and CiP0, candidates of AP endonucleases in Ciona intestinalis, have 3′-5′ exonuclease activity and contribute to protection against oxidative stress

Autor: Masafumi Funakoshi, Daisuke Nambara, Yuichiro Hayashi, Qiu-Mei Zhang-Akiyama

Publikováno v: Genes and Environment, Vol 39, Iss 1, Pp 1-8 (2017)

Abstract Apurinic/apyrimidinic (AP) sites are one of the most frequent DNA lesions. AP sites inhibit transcription and DNA replication, and induce cell death. AP endonucleases are key enzymes in AP site repair. Several types of AP endonucleases have

Externí odkaz: https://doaj.org/article/be9332d85cc1484c9c1ec6a0ac0bf410

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CiAPEX2 and CiP0, candidates of AP endonucleases in Ciona intestinalis, have 3′-5′ exonuclease activity and contribute to protection against oxidative stress

Autor: Daisuke Nambara, Qiu-Mei Zhang-Akiyama, Yuichiro Hayashi, Masafumi Funakoshi

Publikováno v: Genes and Environment, Vol 39, Iss 1, Pp 1-8 (2017)
Genes and Environment

Apurinic/apyrimidinic (AP) sites are one of the most frequent DNA lesions. AP sites inhibit transcription and DNA replication, and induce cell death. AP endonucleases are key enzymes in AP site repair. Several types of AP endonucleases have been repo

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4f0a7f22f1f366ed9a61c27c7f11e1b8
https://doi.org/10.1186/s41021-017-0087-7

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Overexpressed HER2 in NSCLC is a possible therapeutic target of EGFR inhibitors

Autor: Nobuyuki, Ise, Kazuya, Omi, Daisuke, Nambara, Shigeki, Higashiyama, Katsutoshi, Goishi

Publikováno v: Anticancer research. 31(12)

"Oncogene addiction" is a concept in which tumor cells exhibit dependence on certain oncogene(s) for their sustained proliferation and survival, thus providing the rationale for molecular targeted therapies. Cancer cells addicted to epidermal growth

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::491f6d95636ebb22b5370cad2834e408
https://pubmed.ncbi.nlm.nih.gov/22199274

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Abstract A139: Development of EML4-ALK addicted cell models for evaluating sensitivity to ALK inhibitors

Autor: Kazuya Omi, Nobuyuki Ise, Daisuke Nambara, Katsutoshi Goishi

Publikováno v: Molecular Cancer Therapeutics. 10:A139-A139

Background: EML4-ALK fusion tyrosine kinase has been implicated in the pathogenesis of a subset of non-small-cell lung cancers (NSCLCs). The fusion kinase is a rational therapeutic target for addicted cancer cells whose sustained proliferation and su

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::dfdcb8d110a83b24082998b2db3f398e
https://doi.org/10.1158/1535-7163.targ-11-a139

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