Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Dagmara, Wojkowska"'
Autor:
Boguslawa Luzak, Marcin Rozalski, Tomasz Przygodzki, Magdalena Boncler, Dagmara Wojkowska, Marcin Kosmalski, Cezary Watala
Publikováno v:
International Journal of Molecular Sciences; Volume 24; Issue 6; Pages: 5312
Several studies report elevated blood platelet activation and altered platelet count in COVID-19 patients, but the role of the SARS-CoV-2 spike protein in this process remains intriguing. Additionally, there is no data that anti-SARS-CoV-2 neutralizi
Autor:
Cezary Watala, Dagmara Wojkowska, Hassan Kassassir, Kamil Karolczak, Karolina Siewiera, Marcin Braun
Publikováno v:
Aging (Albany NY)
Aging has become a significant risk factor for several diseases, including breast cancer. Platelet activation and platelet-cancer cell aggregate fractions were found to increase with tumor progression in a mouse model of breast cancer. At advanced st
Autor:
Joanna Suraj-Prazmowska, Kamil Karolczak, Stefan Chlopicki, Patrycja Kaczara, Agnieszka Kij, Anna Kurpińska, Agata Malinowska, Cezary Watala, Dagmara Wojkowska, Ivars Kalvins, Kamil Przyborowski, Agnieszka Pelesz
Background Platelet-derived protein disulfide isomerase 1 (PDIA1) regulates thrombus formation, but its role in the regulation of platelet function is not fully understood. Aims The aim of this study was to characterize the role of PDIA1 in human pla
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::692b453cbeda6e37f94314083a57ed16
https://ruj.uj.edu.pl/xmlui/handle/item/279600
https://ruj.uj.edu.pl/xmlui/handle/item/279600
Autor:
Dagmara Wojkowska, Anna Selmi, Marta Stasiak, Kamil Karolczak, Maria Swiatkowska, Marcin Popielarski, Moro O. Salifu, Radoslaw Bednarek, Anna Babinska
Publikováno v:
Breast Cancer Research and Treatment
PurposeTo examine the involvement of the F11R/JAM-A protein in breast cancer metastasis, we utilized the F11R/JAM-A antagonistic peptide 4D (P4D) in experiments of transendothelial migration (TEM) of breast cancer cells.MethodsExperiments were conduc
Autor:
Tomasz Przygodzki, Dawid Polak, Karol Kramkowski, Kamil Karolczak, Nina Wolska, Cezary Watala, Dagmara Wojkowska, Tomasz A. Bonda, Marcin Talar
Publikováno v:
International Journal of Molecular Sciences
Volume 22
Issue 6
International Journal of Molecular Sciences, Vol 22, Iss 3074, p 3074 (2021)
Volume 22
Issue 6
International Journal of Molecular Sciences, Vol 22, Iss 3074, p 3074 (2021)
Blood platelets’ adenosine receptors (AR) are considered to be a new target for the anti-platelet therapy. This idea is based on in vitro studies which show that signaling mediated by these receptors leads to a decreased platelet response to activa
Autor:
M. Pietruczuk, Dominika Książek-Winiarek, Karol Jastrzębski, Krzysztof Jóźwiak, Izabela Jatczak-Pawlik, Dagmara Wojkowska, Andrzej Głąbiński
Publikováno v:
Neurologia i Neurochirurgia Polska. 50:155-162
Migration of inflammatory cells from the blood to the central nervous system (CNS) is crucial for development of multiple sclerosis (MS). Inhibition of this process would allow to control disease activity. The first step confirming this approach woul
Publikováno v:
International Journal of Molecular Sciences; Volume 18; Issue 5; Pages: 1000
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 18, Iss 5, p 1000 (2017)
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 18, Iss 5, p 1000 (2017)
The nature of the interaction between Th17 cells and the blood-brain barrier (BBB) is critical for the development of autoimmune inflammation in the central nervous system (CNS). Tumor necrosis factor alpha (TNF-α) or interleukin 17 (IL-17) stimulat
The nature of the interaction between Th17 cells and the blood-brain barrier (BBB) is critical for the development of autoimmune inflammation in the central nervous system (CNS). TNF-a or IL-17 stimulation is known to enhance the adherence of Th17 ce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4db6eb69bff4bcf0d1bccb36cf4470c4
https://doi.org/10.20944/preprints201703.0094.v1
https://doi.org/10.20944/preprints201703.0094.v1