Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Dae-Gun Seo"'
Autor:
Nguyen Phuong Thao, Seo Young Yang, Sunggun Kim, Sang-Bin Lee, Dae-Gun Seo, Chi Thanh Ma, So-Young Park, Young Ho Kim, Hyun Ok Yang
Publikováno v:
Journal of Natural Products. 83:223-230
Cimicifuga dahurica has traditionally been used as an antipyretic, analgesic, and anti-inflammatory agent and as a treatment for uterine and anal prolapse. This study has investigated the potential beneficial effects of this medicinal plant and its c
Autor:
Sang-Bin, Lee, Seo Young, Yang, Nguyen Phuong, Thao, Dae-Gun, Seo, Sunggun, Kim, Chi Thanh, Ma, So-Young, Park, Young Ho, Kim, Hyun Ok, Yang
Publikováno v:
Journal of natural products. 83(2)
Autor:
Ji Yun Yeo, Sunggun Kim, Dae-Gun Seo, So-Young Park, Jae Eun Lee, Kwang Woo Hwang, Jae-Sun Lee, Na Yeon Kim
Publikováno v:
Molecules
Volume 24
Issue 23
Volume 24
Issue 23
Alzheimer&rsquo
s disease (AD) is a progressive, neurodegenerative brain disorder associated with loss of memory and cognitive function. Beta-amyloid (A&beta
) aggregates, in particular, are known to be highly neurotoxic and lead to neurode
s disease (AD) is a progressive, neurodegenerative brain disorder associated with loss of memory and cognitive function. Beta-amyloid (A&beta
) aggregates, in particular, are known to be highly neurotoxic and lead to neurode
Autor:
Sunggun Kim, Kwang Woo Hwang, So-Young Park, Jae-Sun Lee, Da Kyung Lee, Na Yeon Kim, Dae-Gun Seo
Publikováno v:
Phytomedicine : international journal of phytotherapy and phytopharmacology. 63
Background T helper 17 (Th17) cells, which are differentiated from CD4+ T cells, drive inflammation, leading to autoimmune diseases such as psoriasis, rheumatoid arthritis, and inflammatory bowel disease. Therefore, inhibiting Th17 polarization could
Autor:
Na Yeon Kim, Kwang Woo Hwang, So-Young Park, Da Kyung Lee, Dae-Gun Seo, Jae-Sun Lee, Sunggun Kim
Publikováno v:
Phytomedicine. 68:153188
Publikováno v:
Nutrition research (New York, N.Y.). 36(11)
β-Amyloid (Aβ) is a substance of Alzheimer disease (AD), which is generated via the amyloidogenic pathway from amyloid precursor protein (APP) by β-secretase and γ-secretase. Inhibition of Aβ production is a potential therapeutic approach to AD.