Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Da-Yun Feng"'
Autor:
Jia-hua Zhou, Chao Wang, Di Yang, Ying-xi Wu, Da-yun Feng, Huaizhou Qin, Ju-lei Wang, Ming-hao Wei
Publikováno v:
BMC Neurology, Vol 24, Iss 1, Pp 1-13 (2024)
Abstract Objective The primary objective of this study was to explore the clinical characteristics of apoplectic intratumoral hemorrhage in gliomas and offer insights for improving the diagnosis and treatment of this disease. Methods We analyzed the
Externí odkaz:
https://doaj.org/article/05d82dfa502349a8b9304ed01bf3d0b3
Autor:
Shu-Jiao Li, Hui Liu, Fei-Fei Wu, Da-Yun Feng, Shuai Zhang, Jie Zheng, Lu Wang, Fei Tian, Yan-Ling Yang, Ya-Yun Wang
Publikováno v:
Frontiers in Neuroscience, Vol 17 (2023)
Mitochondrial networks are defined as a continuous matrix lumen, but the morphological feature of neuronal mitochondrial networks is not clear due to the lack of suitable analysis techniques. The aim of the present study is to develop a framework to
Externí odkaz:
https://doaj.org/article/9db65235f08a4ec6a14ac276b9592bde
Autor:
Rui-Xin Yang, Jie Lei, Bo-Dong Wang, Da-Yun Feng, Lu Huang, Yu-Qian Li, Tao Li, Gang Zhu, Chen Li, Fang-Fang Lu, Tie-Jian Nie, Guo-Dong Gao, Li Gao
Publikováno v:
Frontiers in Neurology, Vol 8 (2017)
Oxidative stress and mitochondrial dysfunction play critical roles in ischemia/reperfusion (I/R) injury. DJ-1 is an endogenous antioxidant that attenuates oxidative stress and maintains mitochondrial function, likely acting as a protector of I/R inju
Externí odkaz:
https://doaj.org/article/b99e0a6204d94f6ebdafdca89da9b2c2
Publikováno v:
Neural Plasticity, Vol 2015 (2015)
The mammalian target of rapamycin (mTOR), a serine-threonine protein kinase, integrates extracellular signals, thereby modulating several physiological and pathological processes, including pain. Previous studies have suggested that rapamycin (an mTO
Externí odkaz:
https://doaj.org/article/2edda955313a455a8d5f9a62c1f13547
Publikováno v:
Journal of Neurochemistry. 134:892-903
It has been suggested that propofol can modulate microglial activity and hence may have potential roles against neuroinflammation following brain ischemic insult. However, whether and how propofol can inhibit post-cardiac arrest brain injury via inhi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e7ca0842a00740416a91bf2bb302149d
https://doi.org/10.1111/jnc.13179
https://doi.org/10.1111/jnc.13179
Publikováno v:
Anesthesia and analgesia. 123(1)
The mechanisms underlying general anesthesia-induced neurotoxicity are unclear. Astrocytes have been recognized as important contributors to neuronal development. Until now, the response of the astrocytes to neonatal general anesthetic exposure has b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::620a5feabbd346848ccb753b8e270f96
https://pubmed.ncbi.nlm.nih.gov/27003918
https://pubmed.ncbi.nlm.nih.gov/27003918
Publikováno v:
Neural Plasticity, Vol 2015 (2015)
Neural Plasticity
Neural Plasticity
The mammalian target of rapamycin (mTOR), a serine-threonine protein kinase, integrates extracellular signals, thereby modulating several physiological and pathological processes, including pain. Previous studies have suggested that rapamycin (an mTO
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::24c0f6f77118ca2b103ef4a5887b3fa2
https://doaj.org/article/2edda955313a455a8d5f9a62c1f13547
https://doaj.org/article/2edda955313a455a8d5f9a62c1f13547
Publikováno v:
Anesthesia & Analgesia; Jul2016, Vol. 123 Issue 1, p93-102, 10p