Zobrazeno 1 - 7
of 7
pro vyhledávání: '"D.David Pifer"'
Publikováno v:
Proceedings of the National Academy of Sciences. 78:5180-5184
Factor V, a plasma protein cofactor necessary for optimal conversion of prothrombin to thrombin, is also present in considerable concentration in blood platelets (9.9 units per 10(9) platelets). Subcellular fractionation by two methods has localized
Publikováno v:
Prostaglandins. 21(2)
Prostaglandin I2 (PGI2) is hydrolyzed more slowly in human plasma than in buffer at the same pH; this stabilization appears to be due to binding of PGI2 to albumin. The stability of the antiaggregatory potency of PGI2 is similar in human blood and in
Autor:
Carolyn M. Chesney, Genaro M. A. Palmieri, Barbara Baker, D.David Pifer, Syamal K. Bhattacharya, Tulio E. Bertorini
Publikováno v:
Neurology. 32(10)
Calcium (Ca) and magnesium (Mg) content were determined in muscle of 27 patients with Duchenne muscular dystrophy, 36 with other neuromuscular diseases, and 22 whose muscle biopsy specimens were histochemically normal. Muscle Ca was significantly ele
Autor:
J. Griffin, Airozo D, Irving H. Fox, L. Verling, D.David Pifer, Tulio E. Bertorini, Genaro M. A. Palmieri, Carolyn M. Chesney
Publikováno v:
Neurology. 35(1)
Prompted by the controversy on the efficacy of allopurinol in Duchenne muscular dystrophy and by our observations of an abnormal adenine nucleotide turnover in this disease, we conducted an 18 month, double-blind clinical trial with allopurinol and a
Publikováno v:
Oral Presentations.
Kishimoto et al.(J.B.C.255:2273,1980) have demonstrated a Ca2+ and phospholipid-dependent protein kinase (PK) from various mammalian tissues which is markedly stimulated by the addition of unsaturated diacylglycerol. PAF, an alkyl ether analogue of p
Publikováno v:
Seminars in thrombosis and hemostasis. 11(4)
Defective systemic fibrinolysis and articular persistence of fibrinlike material are well recognized in RA. Perturbation of the major plasma protease inhibitors, A1AT, A2MG, and AT III, was explored in RA, psoriatic arthritis, and Reiter's syndrome.
Publikováno v:
Thrombosis research. 14(2-3)
Removal of the telopeptide regions by pepsin inhibits platelet aggregation and release by monomeric collagen in PRP. This inhibitory effect appears to be due to the inability of pepsin modified collagen to form fibrils in plasma, since fibrils formed