Zobrazeno 1 - 10
of 44
pro vyhledávání: '"D. M. Lublin"'
Publikováno v:
Journal of Virology. 70:5143-5152
Enterovirus 70 (EV70) is a recently emerged human pathogen belonging to the family Picornaviridae. The ability of EV70 to infect a wide variety of nonprimate cell lines in vitro is unique among human enteroviruses. The importance of virus receptors a
Autor:
Richard M. Kaufman, N.A. Clarkson, P.A. Pipkin, Jeffrey W. Almond, David J.A. Evans, Trevor Ward, D M Lublin, P D Minor
Publikováno v:
Journal of Virology. 69:5497-5501
CD55, or decay-accelerating factor (DAF), is a cell surface glycoprotein which regulates complement activity by accelerating the decay of C3/C5 convertases. Recently, we and others have established that this molecule acts as a cellular receptor for e
Publikováno v:
The Journal of Immunology. 152:676-685
Human E express two surface forms of decay-accelerating factor (DAF; CD55). On SDS-PAGE under reducing conditions the major form, DAF-1, migrates as a 70-kDa protein and the minor form, DAF-2, present at < 10% the amount of DAF-1, migrates as a 140-k
Publikováno v:
Molecular and Cellular Biology. 13:6385-6392
Cross-linking of glycosyl-phosphatidylinositol (GPI)-anchored membrane proteins on T cells can trigger cell activation. We and others have shown an association between GPI-anchored proteins and the protein tyrosine kinases (PTKs) p56lck and p59fyn, s
Autor:
D J Thomas, D M Lublin
Publikováno v:
The Journal of Immunology. 150:151-160
Decay accelerating factor (DAF) is a complement regulatory protein that protects host tissue from complement-mediated damage by preventing the assembly and/or promoting the dissociation of C3 and C5 convertases. To identify and analyze the DNA sequen
Publikováno v:
The Journal of Immunology. 149:3535-3541
Decay-accelerating factor (DAF or CD55) is a 70-kDa glycosyl-phosphatidylinositol (GPI)-anchored protein that protects cells from complement-mediated lysis by either preventing the formation of or dissociating C3 convertases. Cross-linking of DAF on
Autor:
K E Coyne, S E Hall, S Thompson, M A Arce, T Kinoshita, T Fujita, D J Anstee, W Rosse, D M Lublin
Publikováno v:
The Journal of Immunology. 149:2906-2913
Decay accelerating factor (DAF, CD55) is a glycophospholipid-anchored membrane protein that protects cells from complement-mediated damage by inhibiting the formation and accelerating the decay of C3/C5 convertases. DAF deletion mutants lacking each
Autor:
D M, Lublin
Publikováno v:
Immunohematology. 21(2)
The antigens of the Cromer blood group system are located on the protein decay-accelerating factor (DAF). This system consists of ten high-prevalence and three low-prevalence antigens; the molecular basis for all of these antigens is a single nucleot
Autor:
H, Lee, S E, Woodman, J A, Engelman, D, Volonté, F, Galbiati, H L, Kaufman, D M, Lublin, M P, Lisanti
Publikováno v:
The Journal of biological chemistry. 276(37)
Caveolin-1 was initially identified as a phosphoprotein in Rous sarcoma virus-transformed cells. Previous studies have shown that caveolin-1 is phosphorylated on tyrosine 14 by c-Src and that lipid modification of c-Src is required for this phosphory
Publikováno v:
Arthritis and rheumatism. 44(1)
Decay-accelerating factor (DAF) is a widely expressed, multifunctional cell surface protein involved in complement regulation and cell signaling. Previous studies have demonstrated that endothelial cell (EC) DAF is up-regulated by tumor necrosis fact