Zobrazeno 1 - 10
of 24
pro vyhledávání: '"D. L. Kleiman de Pisarev"'
Autor:
D. L. Kleiman de Pisarev, Andrea Randi, Noelia Miret, Florencia Chiappini, Laura Alvarez, Lorena Vanesa Zárate, Alejandro Español, María Elena Sales, Claudia Cocca, Carolina Pontillo
Publikováno v:
Toxicology Letters. 295:S89
Autor:
María Jesús Obregón, Raquel Martinez-deMena, S. Hernández, Luis Alvarez, R. Kolliker-Frers, D. L. Kleiman de Pisarev
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
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Treatment of male Wistar rats with hexachlorobenzene (HCB) (1000 mg/kg b.w.) for 3-30 days decreases circulating levels of thyroxine (T4) but does not affect triiodothyronine (T3). Time courses were determined for 5′ deiodinase type I (5′ D-I) ac
Autor:
Laura Alvarez, Mario A. Pisarev, P. Hockl, Andrea Randi, S. Hernandez, Adriana Laura Burgueño, S. Zeni, D. L. Kleiman De Pisarev
Publikováno v:
Hormone and Metabolic Research. 36:291-297
3,5,3'-triiodothyroacetic acid (TRIAC) has been used to suppress pituitary TSH secretion with reported attenuation of extrapituitary effects. We investigated whether equivalent doses of T (3) and TRIAC preventing the induction of goiter by methimazol
Autor:
A M Ferramola de Sancovich, Pilar Santisteban, H A Sancovich, Andrea Randi, A I Loaiza-Pérez, M Seisdedos, D. L. Kleiman de Pisarev
Publikováno v:
Endocrinology. 140:4142-4151
Hexachlorobenzene (HCB) is a dioxin-type chemical that acts mainly through the aryl hydrocarbon receptor. Chronic exposure of rats to HCB increases the activity of malic enzyme (ME). In this report, we show that this increase is correlated with an in
Autor:
Claudia Cocca, Andrea Randi, Carolina Pontillo, Noelia Miret, Laura Alvarez, Florencia Chiappini, Alejandro Español, D. L. Kleiman de Pisarev, María Elena Sales, L. Marcucci
Publikováno v:
Toxicology Letters. 259:S94
Autor:
A I, Loaiza-Pérez, M T, Seisdedos, D L, Kleiman de Pisarev, H A, Sancovich, A S, Randi, A M, Ferramola de Sancovich, P, Santisteban
Publikováno v:
Endocrinology. 140(9)
Hexachlorobenzene (HCB) is a dioxin-type chemical that acts mainly through the aryl hydrocarbon receptor. Chronic exposure of rats to HCB increases the activity of malic enzyme (ME). In this report, we show that this increase is correlated with an in
Publikováno v:
Journal of endocrinological investigation. 22(6)
The effect of the in vivo administration of hexachlorobenzene (HCB) (100 mg/100 g bw) for 30 days, on the activities of brown adipose tissue (BAT) lipogenic enzymes, i.e. malic enzyme (ME), and glucose-6-phosphate dehydrogenase (G6PD) and the mitocho
Autor:
A I, Loaiza Perez, H A, Sancovich, D L, Kleiman De Pisarev, A S, Randi, M, Seisdedos, A M, Ferramola De Sancovich, P, Santisteban
Publikováno v:
Acta physiologica, pharmacologica et therapeutica latinoamericana : organo de la Asociacion Latinoamericana de Ciencias Fisiologicas y [de] la Asociacion Latinoamericana de Farmacologia. 48(3)
Hexachlorobenzene (HCB) is a widespread environmental pollutant. Chronic exposure of laboratory animals to HCB triggers porphyria, induction of liver microsomal enzymes, low levels of T4 reproductive dysfunction's, liver and thyroid tumors. Previous
Publikováno v:
Journal of endocrinological investigation. 18(4)
The functional thyroid status of hexachlorobenzene (HCB)-treated rats was studied. HCB caused a depletion of serum thyroxine (T4 ), but did not change L-3,5,3’-triiodothyronine (T3) levels in serum of rats. The activities of the thyroid regulated m
Publikováno v:
Journal of endocrinological investigation. 17(5)
The role of thyroid status in hexachlorobenzene (HBC) induced porphyria was studied in normal, thyroidectomized and thyroxine (T4) treated rats. Female Wistar rats were treated with HCB for different periods of time. Serum T4 levels were depressed af