Zobrazeno 1 - 10
of 763
pro vyhledávání: '"D. Heinegård"'
Autor:
D, Heinegård
Publikováno v:
Annals of the rheumatic diseases. 34
Publikováno v:
Clinical Orthopaedics and Related Research. 330:251-260
In situ hybridization and ultrastructural immunohistochemistry were used to study the synthesis and distribution of 2 bone matrix proteins, bone sialoprotein and osteopontin, in the proximal tibial epiphysis in 21-, 32-, and 84-day-old rats. Bone sia
Publikováno v:
Journal of Biological Chemistry. 269:21547-21554
A leucine-rich protein, chondroadherin, has been isolated from dissociative extracts of articular cartilage, and its primary structure has been determined by both direct protein sequencing and DNA sequence analysis of polymerase chain reaction produc
Publikováno v:
Journal of Biological Chemistry. 269:14853-14856
The tartrate-resistant acid phosphatase (TRAP) of skeletal osteoclasts was found to partially dephosphorylate the bone matrix phosphoproteins osteopontin (OPN) and bone sialoprotein (BSP). TRAP also partially dephosphorylated metabolically [32P]PO4-l
Autor:
Alberto A Palacios-Boix, D. Heinegård, Edwin Choy, P Jobanputra, Gabriel S. Panayi, Gabrielle H. Kingsley, J. Sieper
Publikováno v:
Annals of the Rheumatic Diseases. 51:959-962
Cellular immunity to cartilage proteoglycans may be responsible for sustaining chronic inflammation in ankylosing spondylitis. This hypothesis was examined by measuring peripheral blood and synovial fluid mononuclear cell proliferation in five prepar
Publikováno v:
Biorheology. 41(5)
The effect of strain and transforming growth factor beta on equine tendon fibroblasts (tenocytes) was assessed in vitro. Tenocytes were isolated from flexor and extensor tendons of horses from foetal to 10 years of age. These cells were cultured unti
Publikováno v:
Histology and histopathology. 19(2)
During accelerated vascular remodeling such as in atherosclerosis, the composition of the extracellular matrix becomes altered. The matrix components of the diseased artery influence cellular processes such as adhesion, migration and proliferation. F
Autor:
D. Heinegård
Publikováno v:
Osteoarthritis and Cartilage. 17:S2-S3
Autor:
L A, Joosten, E, Lubberts, M M, Helsen, T, Saxne, C J, Coenen-de Roo, D, Heinegård, W B, van den Berg
Publikováno v:
Arthritis Research
Destruction of cartilage and bone are hallmarks of human rheumatoid arthritis (RA), and controlling these erosive processes is the most challenging objective in the treatment of RA. Systemic interleukin-4 treatment of established murine collagen-indu
Autor:
J F, Bleasel, A R, Poole, D, Heinegård, T, Saxne, D, Holderbaum, M, Ionescu, P, Jones, R W, Moskowitz
Publikováno v:
Arthritis and rheumatism. 42(1)
The Arg5l9-Cys mutation in type II collagen results in severe, precocious familial osteoarthritis (OA) in 100% of carriers within the first 3 decades of life. The carrier population provided a well-defined patient population for the study of serum ma