Zobrazeno 1 - 10
of 106
pro vyhledávání: '"D. A. Dahl"'
Autor:
Matías J. Dahl, Kristopher A. Lofgren, Cleo Haugen, Gil E. Harmon, Sarah P. Hughes, Karen D. Cowden Dahl
Publikováno v:
Frontiers in Oncology, Vol 14 (2024)
Uterine carcinosarcoma (UCS) is a rare but aggressive endometrial cancer. Survival outcomes for women diagnosed with UCS remain poor with lower survival than those of endometrioid or high-grade serous uterine cancers. The histopathological hallmark o
Externí odkaz:
https://doaj.org/article/a5d87c31f11b475ebae3d13f1d624031
Publikováno v:
Cancers, Vol 13, Iss 2, p 274 (2021)
Ovarian cancer (OC) cells survive in the peritoneal cavity in a complex microenvironment composed of diverse cell types. The interaction between tumor cells and non-malignant cells is crucial to the success of the metastatic process. Macrophages acti
Externí odkaz:
https://doaj.org/article/3aa7afb4152d4ca6a039e6c0704d55c3
Autor:
Andrew Appert, Sridhar Rao, Seok Hee Jang, Richard Dahl, Nathan Klopfenstein, William Morgan Hallas, Kirthi Pulakanti, Karen D. Cowden Dahl, Austin Boucher, Jennifer Skibbe
Publikováno v:
J Immunol
Macrophages are critical for regulating inflammatory responses. Environmental signals polarize macrophages to either a proinflammatory (M1) state or an anti-inflammatory (M2) state. We observed that the microRNA (miRNA) cluster mirn23a, coding for mi
Autor:
Jeffrey L Kurkewich, Nathan Klopfenstein, William M Hallas, Christian Wood, Rachel A Sattler, Chhaya Das, Haley Tucker, Richard Dahl, Karen D Cowden Dahl
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0161468 (2016)
Arid3a and Arid3b belong to a subfamily of ARID (AT-rich interaction domain) transcription factors. The Arid family is involved in regulating chromatin accessibility, proliferation, and differentiation. Arid3a and Arid3b are closely related and share
Externí odkaz:
https://doaj.org/article/5ceb529aeaee474cbd4dc9258760b72b
Autor:
Lynn Roy, Karen D. Cowden Dahl
Publikováno v:
Cancers, Vol 10, Iss 8, p 241 (2018)
Ovarian cancer is the most lethal gynecological malignancy. Poor overall survival, particularly for patients with high grade serous (HGS) ovarian cancer, is often attributed to late stage at diagnosis and relapse following chemotherapy. HGS ovarian c
Externí odkaz:
https://doaj.org/article/0a180ec91ff4417cad153b94a21f5c91
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 11, Iss 11, Pp 1208-1215 (2009)
The epithelial-to-mesenchymal transition (EMT) that occurs during embryonic development is recapitulated during tumor metastasis. Important regulators of this process include growth factors, transcription factors, and adhesion molecules. New evidence
Externí odkaz:
https://doaj.org/article/04a320d3f3c5492f87664bc45244ce84
Autor:
Alexander Bobbs, Katrina Gellerman, William Morgan Hallas, Stancy Joseph, Chao Yang, Jeffrey Kurkewich, Karen D Cowden Dahl
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0131961 (2015)
The DNA-binding protein AT-Rich Interactive Domain 3B (ARID3B) is elevated in ovarian cancer and increases tumor growth in a xenograft model of ovarian cancer. However, relatively little is known about ARID3B's function. In this study we perform the
Externí odkaz:
https://doaj.org/article/a1912aa8f40c40d98baf5973ac6f2017
Autor:
Karen D. Cowden Dahl
Publikováno v:
Current Drug Targets. 23:769-769
Publikováno v:
Cancers
Volume 13
Issue 2
Cancers, Vol 13, Iss 274, p 274 (2021)
Volume 13
Issue 2
Cancers, Vol 13, Iss 274, p 274 (2021)
Simple Summary Ovarian cancer is the 5th leading cause of cancer death in US women, due to late diagnosis. The vast majority of patients with ovarian cancer have metastatic disease at diagnosis, leading to poor survival. As the tumor cells metastasiz
Publikováno v:
PLoS ONE, Vol 7, Iss 7, p e42159 (2012)
Alternative splicing is a common occurrence in many cancers. Alternative splicing is linked with decreased apoptosis and chemoresistance in cancer cells. We previously demonstrated that ARID3B, a member of the AT-rich interactive domain (ARID) family
Externí odkaz:
https://doaj.org/article/cd0d3e2718624f04a52f7c98540b84b0