Zobrazeno 1 - 10 of 23 pro vyhledávání: '"D V Serreze"'
1
Unexpected functional consequences of xenogeneic transgene expression in ?-cells of NOD mice
Autor: Peter C. Reifsnyder, Edward H. Leiter, J. Driver, Alexander V. Chervonsky, S. Kamdar, Caroline Morgane Choisy-Rossi, M. Hara, D. V. Serreze
Publikováno v: Diabetes, Obesity and Metabolism. 9:14-22
We describe unexpected alterations in the non-obese diabetic (NOD/Lt) mouse model of type 1 diabetes (T1D) following forced beta-cell expression of non-mammalian genes ligated to an insulin promoter sequence. These include the jellyfish green fluores
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe4b686120119e044e4281930fbcda33
https://doi.org/10.1111/j.1463-1326.2007.00770.x
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2
Initiation of autoimmune diabetes in NOD/Lt mice is MHC class I-dependent
Autor: D V Serreze, H D Chapman, D S Varnum, I Gerling, E H Leiter, L D Shultz
Publikováno v: The Journal of Immunology. 158:3978-3986
MHC class II alleles clearly contribute a primary genetic component of susceptibility to autoimmune insulin-dependent diabetes mellitus (IDDM) in nonobese diabetic (NOD) mice. However, IDDM does not occur in NOD mice made MHC class I-deficient by a f
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::288ddd57cf95152a1cd54929bd6e5c0a
https://doi.org/10.4049/jimmunol.158.8.3978
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8
Development of diabetogenic T cells from NOD/Lt marrow is blocked when an allo-H-2 haplotype is expressed on cells of hemopoietic origin, but not on thymic epithelium
Autor: D V Serreze, E H Leiter
Publikováno v: The Journal of Immunology. 147:1222-1229
Diabetes is a T cell-mediated process in NOD/Lt mice, with a major genetically recessive component of susceptibility linked to homozygous expression of the unique H-2g7 MHC haplotype. Heterozygous expression of the H-2nb1 haplotype derived from the N
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::0498cf6ddf2e206389e75ef62f975f6a
https://doi.org/10.4049/jimmunol.147.4.1222
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10
B lymphocytes are critical antigen-presenting cells for the initiation of T cell-mediated autoimmune diabetes in nonobese diabetic mice
Autor: D V, Serreze, S A, Fleming, H D, Chapman, S D, Richard, E H, Leiter, R M, Tisch
Publikováno v: Journal of immunology (Baltimore, Md. : 1950). 161(8)
Nonobese diabetic (NOD) mice genetically deficient in B lymphocytes (NODJg mu(null)) are resistant to T cell-mediated autoimmune insulin-dependent diabetes mellitus (IDDM). Ig infusions from diabetic NOD donors did not abrogate IDDM resistance in NOD
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::1e8d26cc1bb4b5347bd766ab76442285
https://pubmed.ncbi.nlm.nih.gov/9780157
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