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Publikováno v:
Brain research. 861(2)
Glutamate-mediated excitotoxicity has been shown to contribute to cellular dysfunction following traumatic brain injury (TBI). Increasing inhibitory function through stimulation of gamma-aminobutyric acid (GABA(A)) receptors may attenuate excitotoxic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c46498cdce35f16f161633154a5eb1bd
https://pubmed.ncbi.nlm.nih.gov/10760494
https://pubmed.ncbi.nlm.nih.gov/10760494
