Zobrazeno 1 - 10
of 13
pro vyhledávání: '"D M, Kullmann"'
Autor:
S M, Zuberi, L H, Eunson, A, Spauschus, R, De Silva, J, Tolmie, N W, Wood, R C, McWilliam, J B, Stephenson, J P, Stephenson, D M, Kullmann, M G, Hanna
Publikováno v:
Brain. 122:817-825
Episodic ataxia type 1 (EA1) is a rare autosomal dominant disorder characterized by brief episodes of ataxia associated with continuous interattack myokymia. Point mutations in the human voltage-gated potassium channel (Kv1.1) gene on chromosome 12p1
Publikováno v:
BMJ (Clinical research ed.). 345
Autor:
R. C. Wykes, J. H. Heeroma, L. Mantoan, K. Zheng, D. C. MacDonald, K. Deisseroth, K. S. Hashemi, M. C. Walker, S. Schorge, D. M. Kullmann
Publikováno v:
Science Translational Medicine.
Autor:
D M, Kullmann
Publikováno v:
Neuron. 32(4)
Kainate receptor agonists depress transmitter release at several synapses in the hippocampus. Distinct mechanisms appear to underlie this phenomenon at different synapses. Recently, it has emerged that presynaptic kainate receptors can also potentiat
Publikováno v:
Neurology. 74:269-270
Autor:
D M, Kullmann
Publikováno v:
Progress in brain research. 125
Autor:
L H, Eunson, R, Rea, S M, Zuberi, S, Youroukos, C P, Panayiotopoulos, R, Liguori, P, Avoni, R C, McWilliam, J B, Stephenson, M G, Hanna, D M, Kullmann, A, Spauschus
Publikováno v:
Annals of neurology. 48(4)
Episodic ataxia type 1 (EA1) is an autosomal dominant central nervous system potassium channelopathy characterized by brief attacks of cerebellar ataxia and continuous interictal myokymia. Point mutations in the voltage-gated potassium channel gene K
Publikováno v:
Neuron. 21(3)
Glutamatergic transmission at mossy fiber (MF) synapses on CA3 pyramidal neurons in the hippocampus is mediated by AMPA, kainate, and NMDA receptors and undergoes presynaptic modulation by metabotropic glutamate receptors. The recruitment of differen
Autor:
D M, Kullmann
Publikováno v:
Neuron. 12(5)
Quantal analysis has provided evidence for a presynaptic contribution to long-term potentiation in hippocampal CA1 cells. This however leaves unexplained the observation that long-term potentiation has little or no effect on the NMDA receptor-mediate
Publikováno v:
Neurology. 76:490-490