Zobrazeno 1 - 10
of 28
pro vyhledávání: '"D J, Salant"'
Autor:
R J, Quigg, Y, Kozono, D, Berthiaume, A, Lim, D J, Salant, A, Weinfeld, P, Griffin, E, Kremmer, V M, Holers
Publikováno v:
The Journal of Immunology. 160:4553-4560
A recombinant soluble form of the mouse membrane complement inhibitor Crry (complement receptor-related gene y) fused to IgG1 hinge, CH2, and CH3 domains has been created and designated Crry-Ig. Crry has been used because, similar to human soluble CR
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7aec6b8ecd1f8e8aca9d379bde01a28d
https://doi.org/10.4049/jimmunol.160.9.4553
https://doi.org/10.4049/jimmunol.160.9.4553
Publikováno v:
Experimental Nephrology. 4(3):184-192
Cultures of glomerular epithelial cells (GEC) are currently used to identify important cellular and molecular mechanisms involved in the pathogenesis of renal diseases. However, there is still controversy in the literature as to the visceral or parie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::dec29740329ad3ee59fa61baeb820c43
https://www.ncbi.nlm.nih.gov/pubmed/8773481
https://www.ncbi.nlm.nih.gov/pubmed/8773481
Publikováno v:
The Journal of Immunology. 147:838-845
In passive Heymann nephritis, a rat model of membranous nephropathy, antibody (anti-Fx1A) activates C on the surface of the glomerular epithelial cell (GEC), leading to GEC injury and proteinuria. In this study, we examined C activation by anti-Fx1A
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c053583a36dde657240935f34cfe8a14
https://doi.org/10.4049/jimmunol.147.3.838
https://doi.org/10.4049/jimmunol.147.3.838
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 147(3)
In passive Heymann nephritis, a rat model of membranous nephropathy, antibody (anti-Fx1A) activates C on the surface of the glomerular epithelial cell (GEC), leading to GEC injury and proteinuria. In this study, we examined C activation by anti-Fx1A
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8265f64fa1ea98f93f33d2b0f0b2631e
https://pubmed.ncbi.nlm.nih.gov/1861077
https://pubmed.ncbi.nlm.nih.gov/1861077
Publikováno v:
The American journal of pathology. 138(2)
The distribution and synthetic rate of glomerular basement membrane components was examined in the Passive Heymann Nephritis model of experimental membranous nephropathy. The extensive tissue injury that developed included subepithelial electron-dens
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::bc8e08f09b5b8f66497bfde2c95d17cf
https://pubmed.ncbi.nlm.nih.gov/1992771
https://pubmed.ncbi.nlm.nih.gov/1992771
Publikováno v:
The American journal of pathology. 137(1)
Many earlier studies have shown that the intravenous injection into rats of sheep antibodies against rat glomerular basement membrane (GBM) induces a rapid influx of neutrophils and proteinuria (nephrotoxic nephritis or NTN). The GBM antigens recogni
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::500de049b12e4d71a5ceb5b1ae6d5466
https://pubmed.ncbi.nlm.nih.gov/2196815
https://pubmed.ncbi.nlm.nih.gov/2196815
Publikováno v:
The Journal of Immunology. 142:877-882
Epithelial cells of the glomerular capillary are the site of C5b-9 mediated injury in rat membranous nephropathy. We investigated the regulation of C activation by cultured glomerular epithelial cells (GEC). Rat and human GEC were more resistant to C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3355b6384cc40bbed3d482deb1c44aac
https://doi.org/10.4049/jimmunol.142.3.877
https://doi.org/10.4049/jimmunol.142.3.877
Publikováno v:
The Journal of Immunology. 137:1511-1516
Using a model of rat membranous nephropathy (MN), we examined the relationship between the development of glomerular epithelial cell injury and the formation and stability of the membrane attack complex (MAC) of complement. Isolated rat kidneys were
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a4a26b346b67e7527702e8ea0028a966
https://doi.org/10.4049/jimmunol.137.5.1511
https://doi.org/10.4049/jimmunol.137.5.1511
Autor:
D J, Salant, R, Rabkin
Publikováno v:
South African medical journal = Suid-Afrikaanse tydskrif vir geneeskunde. 49(37)
A patient with primary hyperparathyroidism who presented with hypokalaemia and hypertension is described. Renal potassium wasting was documented and cured by removal of a parathyroid adenoma. Possible mechanisms for this unusual manifestation of hype
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2c934d817abfb0c0b77a7ca5de9b4e57
https://pubmed.ncbi.nlm.nih.gov/1162532
https://pubmed.ncbi.nlm.nih.gov/1162532
Publikováno v:
The American journal of pathology. 129(2)
Binding of anti-Fx1A to Heymann nephritis antigens (HA) on rat glomerular epithelial cells (GECs) in culture leads to capping and disappearance of antigens from the cell surface. This process may contribute to the formation of glomerular subepithelia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7ce7781bf1d4af0b5e42d7444b604461
https://pubmed.ncbi.nlm.nih.gov/3674205
https://pubmed.ncbi.nlm.nih.gov/3674205