Zobrazeno 1 - 10
of 84
pro vyhledávání: '"D DeMaria"'
Publikováno v:
Plant Protection Science, Vol 38, Iss SI 2 - 6th Conf EFPP, Pp 490-493 (2002)
In order to investigate cross-protection mechanisms between stresses of different origins, greenhouse experiments were conducted to determine whether resistance levels to the fungal pathogen P. capsici were affected on wounded plants. To this purpose
Externí odkaz:
https://doaj.org/article/933fa038760a4c3ba22fd24e7edd49cf
Akademický článek
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Publikováno v:
Fisheries Research. 99:123-129
We examined sagittal otoliths from 398 gray angelfish (Pomacanthus arcuatus) collected from the lower Florida Keys between September 2000 and September 2003. Fish ranged in size from 78 to 442 mm total length (TL). Males had a mean TL of 329 mm (n =
Autor:
Carla D. DeMaria, Siao Yun Chang, Dipayan Chaudhuri, Rebecca S. Alvania, David T. Yue, Tuck Wah Soong
Publikováno v:
The Journal of Neuroscience. 24:6334-6342
Alternative splicing of the P/Q-type channel (CaV2.1) promises customization of the computational repertoire of neurons. Here we report that concerted splicing of its main α1Asubunit, at both an EF-hand-like domain and the channel C terminus, contro
Publikováno v:
Proceedings of the National Academy of Sciences. 99:17185-17190
Engineered calmodulins (CaMs), rendered Ca 2+ -insensitive by mutations, function as dominant negatives in heterologous systems, and have revealed mechanisms of ion channel modulation by Ca 2+ /CaM. The use of these CaMs in native mammalian cells now
Autor:
Rebecca S. Alvania, Carla D. DeMaria, Scott Mittman, William S. Agnew, David T. Yue, Larry S. Zweifel, Tuck Wah Soong, Mui Cheng Liang
Publikováno v:
The Journal of Neuroscience. 22:10142-10152
P/Q-type (Ca(v)2.1) calcium channels support a host of Ca2+-driven neuronal functions in the mammalian brain. Alternative splicing of the main alpha1A (alpha1(2.1)) subunit of these channels may thereby represent a rich strategy for tuning the functi
Publikováno v:
Nature. 411:484-489
Acute modulation of P/Q-type (α1A) calcium channels by neuronal activity-dependent changes in intracellular Ca2+ concentration may contribute to short-term synaptic plasticity1,2,3, potentially enriching the neurocomputational capabilities of the br
Autor:
Blaise Z. Peterson, Trudy A. Kohout, Terry B. Rogers, Shao Kui Wei, David T. Yue, Rui Zhang, Henry M. Colecraft, Carla D. DeMaria
Publikováno v:
Circulation Research. 86:175-184
Abstract —L-type Ca 2+ channels contribute importantly to the normal excitation-contraction coupling of physiological hearts, and to the functional derangement seen in heart failure. Although Ca 2+ channel auxiliary β 1–4 subunits are among the
Autor:
J. Sludden, P. Wissel, D DeMaria, Chris Twelves, Stan B. Kaye, A. McDonald, Peter J. O'Dwyer, Louise B. Grochow, James P. Stevenson, K. Selinger, L. Paz-Ares
Publikováno v:
Annals of Oncology. 10:339-344
Summary Background Preclinical results support a prolonged schedule of administration for topoisomerase I inhibitors, and we have previously demonstrated the safety and activity of the novel water-soluble topoisomerase I inhibitor GG211 when given as
Publikováno v:
Neuron. 22(3):549-558
Elevated intracellular Ca2+ triggers inactivation of L-type calcium channels, providing negative Ca2+ feedback in many cells. Ca2+ binding to the main alpha1c channel subunit has been widely proposed to initiate such Ca2+ -dependent inactivation. Her