Zobrazeno 1 - 10
of 66
pro vyhledávání: '"D C Allred"'
Autor:
Rodrigo Franco Gonçalves, Souzan Sanati, Yu Tao, Maggie C.U. Cheang, D. C. Allred, Erika C. Crouch, Michael Barnes, Katherine DeSchryver, Jeremy Hoog, Matthew J. Ellis, John Olson, Cynthia X. Ma, Luis Octavio Zanatta Sarian, Neha Dahiya
Publikováno v:
Breast Cancer Research and Treatment
Purpose The recent publication of the ACOSOG Z1031 trial results demonstrated that Ki-67 proliferation marker-based neoadjuvant endocrine therapy response monitoring could be used for tailoring the use of adjuvant chemotherapy in ER+HER2-negative bre
Autor:
J. Shou, Lesley-Ann Martin, D. C. Allred, Janine Salter, Ian E. Smith, Simone Detre, Mitch Dowsett, Rachel Schiff, Margaret Hills, CK Osborne, Gutierrez Mc, Syed K. Mohsin, S.R.D. Johnston
Publikováno v:
Endocrine-Related Cancer. 12:S113-S117
Growth factor signalling and response to endocrine therapy: the Royal Marsden Experience De novo resistance to endocrine therapy is a near-universal feature of oestrogen receptor (ER)negative breast cancer. Although many ER-positive breast cancers al
Autor:
D. C. Allred, Cynthia X. Ma, Erika C. Crouch, Yu Tao, Vera J. Suman, Rodrigo Franco Gonçalves, S Sanati, Katherine DeSchryver, MJ Ellis, Michael Barnes
Publikováno v:
Cancer Research. 73:P3-05
Ki-67 proliferation marker-based response monitoring will be prospectively tested in the ALternate Treatment for ER positive NeoAdjuvant TrEatment (ALTERNATE) trial. Tumor Ki-67 will be analyzed after one month of neoadjuvant anastrozole, fulvestrant
Autor:
S.G. Bonnette, Darryl L. Hadsell, Adrian V. Lee, Ping Zhang, Jeffrey M. Rosen, Russell C. Hovey, Barbara K. Vonderhaar, C R Kahn, Jessy George, D. C. Allred, Sandra L. Grimm, Syed K. Mohsin, Margarita M. Ivanova, Daniel Torres, Steffi Oesterreich
Publikováno v:
Endocrinology. 144:2683-2694
Insulin receptor substrates (IRS) are central integrators of hormone, cytokine, and growth factor signaling. IRS proteins can be phosphorylated by a number of signaling pathways critical to normal mammary gland development. Studies in transgenic mice
Autor:
Jessica M. Archambault, Szeman Ruby Chan, Kathleen C. F. Sheehan, Julie A. Allen, Silvia Lonardi, Theresa M. McDevitt, J M White, Cora D. Arthur, William Vermi, Robert D. Schreiber, Deepta Bhattacharya, Charles G. Rickert, M V Lorenzi, D C Allred
We previously reported that STAT1 expression is frequently abrogated in human estrogen receptor-α-positive (ERα+) breast cancers and mice lacking STAT1 spontaneously develop ERα+ mammary tumors. However, the precise mechanism by which STAT1 suppre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a9fca8fafb59a65f9294e6d6e046170
http://hdl.handle.net/11379/454746
http://hdl.handle.net/11379/454746
Publikováno v:
Endocrine-related cancer. :47-61
Most human invasive breast cancers (IBCs) appear to develop over long periods of time from certain pre-existing benign lesions. Of the many types of benign lesions in the human breast, only a few appear to have significant premalignant potential. The
Autor:
D C Allred, S K Mohsin
Publikováno v:
Journal of Mammary Gland Biology and Neoplasia. 5:351-364
Most human invasive breast cancers (IBCs) arise from preexisting benign lesions. There are many types of benign lesions in the human breast and only a few appear to have significant premalignant potential (atypical hyperplasias and in situ carcinomas
Apoptosis and proliferation as predictors of chemotherapy response in patients with breast carcinoma
Publikováno v:
Cancer. 89:2145-2152
BACKGROUND Laboratory evidence suggests that many anticancer agents exert their effect by altering the ratio between apoptosis and cellular proliferation. The objective of this clinical study was to examine in vivo changes in apoptotic index (AI), Bc
Autor:
D. C. Allred, Syed K. Mohsin, SG Hilsenbeck, Suzanne A. W. Fuqua, Gary M. Clark, CK Osborne, Peter O'Connell, K. Fischbach
Publikováno v:
JNCI Journal of the National Cancer Institute. 91:1391-1397
Background: In breast cancer progression, the prevalence of damage at specific genetic loci often increases with the stage of the lesion (i.e., from noninvasive to invasive to metastatic). By use of genetic markers and analysis of allelic imbalances
Autor:
Grazia Arpino, Francisco E. Gago, L M Vargas-Roig, Syed K. Mohsin, Heidi L. Weiss, Richard M. Elledge, Daniel R. Ciocca, M. Leuzzi, D. C. Allred, P. Daguerre
Publikováno v:
e_Buah Biblioteca Digital Universidad de Alcalá
instname
instname
Purpose. Laboratory evidence indicates that tumor growth depends on the balance between cell proliferation and cell death, and many anticancer agents may exert their therapeutic effect by decreasing proliferation and increasing apoptosis. Additionall
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3459a3d7d9b05a1072655200cfcabd82
https://hdl.handle.net/10017/5043
https://hdl.handle.net/10017/5043