Zobrazeno 1 - 10
of 54
pro vyhledávání: '"Cynthia St Hilaire"'
Autor:
Brandon T. Garland, MD, Manfred Boehm, MD, Peter C. Grayson, MD, Cynthia St Hilaire, PhD, Alessandra Brofferio, MD, Benjamin W. Starnes, MD
Publikováno v:
Journal of Vascular Surgery Cases and Innovative Techniques, Vol 2, Iss 4, Pp 190-192 (2016)
Takayasu arteritis is a large-vessel vasculitis that often results in pulselessness due to fibrotic stenoses. Whereas minor calcification is sometimes seen with Takayasu arteritis, it rarely causes stenosis. Extensive calcification resulting in malpe
Externí odkaz:
https://doaj.org/article/4d0a260a221a4555a4f1e3c49947d192
Autor:
Rachel G. Kroll, Corey Powell, Jun Chen, Natasha T. Snider, Cynthia St. Hilaire, Akshay Reddy, Judy Kim, David J. Pinsky, Venkatesh L. Murthy, Nadia R. Sutton
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 12, Iss 9 (2023)
Background Ectonucleotidases maintain vascular homeostasis by metabolizing extracellular nucleotides, modulating inflammation and thrombosis, and potentially, myocardial flow through adenosine generation. Evidence implicates dysfunction or deficiency
Externí odkaz:
https://doaj.org/article/9cb0e3342def42b2ac15ae5b602fd5ba
Autor:
Rolando A. Cuevas, Ryan Wong, Pouya Joolharzadeh, William J. Moorhead, Claire C. Chu, Jack Callahan, Alex Crane, Camille K. Boufford, Angelina M. Parise, Aneesha Parwal, Parya Behzadi, Cynthia St. Hilaire
Publikováno v:
American Journal of Physiology-Cell Physiology. 324:C327-C338
Arterial calcification due to deficiency of CD73 (ACDC) is a rare genetic disease caused by a loss-of-function mutation in the NT5E gene encoding the ecto-5′-nucleotidase (cluster of differentiation 73, CD73) enzyme. Patients with ACDC develop vess
Autor:
Rolando A. Cuevas, Cynthia St. Hilaire
Publikováno v:
Circulation research. 130(10)
Autor:
Cynthia St. Hilaire
Publikováno v:
Arterioscler Thromb Vasc Biol
Over 200 million individuals worldwide are estimated to have peripheral artery disease (PAD). Although the term peripheral can refer to any outer branch of the vasculature, the focus of this review is on lower-extremity arteries. The initial sequelae
Autor:
Fadil M. Hannan, Cynthia St. Hilaire, Ángel Millán, Yu Sato, Renu Virmani, Mirjam Schuchardt, Paolo Raggi, Roberto Ferraresi, Dominic Furniss, Peter Lanzer, Pak-Wing Fok, Jan Janzen, Rajesh V. Thakker, Julio Saez-Rodriguez, Jan D. Lanzer
Publikováno v:
Journal of the American College of Cardiology. 78:1145-1165
Medial arterial calcification (MAC) is a chronic systemic vascular disorder distinct from atherosclerosis that is frequently but not always associated with diabetes mellitus, chronic kidne...
Autor:
Nadia R. Sutton, Rajeev Malhotra, Cynthia St. Hilaire, Elena Aikawa, Roger S. Blumenthal, Grace Gackenbach, Parag Goyal, Adam Johnson, Sagar U. Nigwekar, Catherine M. Shanahan, Dwight A. Towler, Brooke N. Wolford, Yabing Chen
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 43(1)
Cardiovascular disease is the most common cause of death worldwide, especially beyond the age of 65 years, with the vast majority of morbidity and mortality due to myocardial infarction and stroke. Vascular pathology stems from a combination of genet
Autor:
Scott A. Hahn, Ying Tang, Mark T. Gladwin, Taijyu Satoh, Charles F. McTiernan, Kentaro Noda, Cynthia St. Hilaire, Bing Wang, Adam C. Straub, Samuel K. Wyman, Cristina Espinosa-Diez, Georgios Triantafyllou, Jeffrey J. Baust, Sruti Shiva, Matthew R Dent, Longfei Wang, Yijen L. Wu, Elena A. Goncharova, Dmitry A. Goncharov, Mike Reynolds, Yen Chun Lai, Andrea R. Levine, Elizabeth R. Rochon, Delphine Gomez, Stephen Y. Chan
Publikováno v:
Circulation
Circulation, vol 144, iss 8
Circulation, vol 144, iss 8
Background: Many patients with heart failure with preserved ejection fraction have metabolic syndrome and develop exercise-induced pulmonary hypertension (EIPH). Increases in pulmonary vascular resistance in patients with heart failure with preserved
Autor:
Cynthia St. Hilaire
Publikováno v:
Circulation Research. 132:481-482
Autor:
Rolando A. Cuevas, Luis Hortells, Claire C. Chu, Ryan Wong, Alex Crane, Camille Boufford, Cailyn Regan, William J. Moorhead, Michael J. Bashline, Aneesha Parwal, Angelina M. Parise, Aditi Gurkar, Dennis Bruemmer, John Sembrat, Ibrahim Sultan, Thomas G. Gleason, Marie Billaud, Cynthia St. Hilaire
BackgroundCalcific aortic valve disease (CAVD) is the pathological remodeling of the valve leaflets which leads to heart failure and high stroke risk. While several mechanisms are known to drive cardiovascular calcification, the initial steps orchest
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2d9099013df050e8bad11c75355c1c25
https://doi.org/10.1101/2022.08.23.504425
https://doi.org/10.1101/2022.08.23.504425