Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Cynthia Soderblom"'
Autor:
Do-Hun Lee, Kara L. Spiller, Cynthia Soderblom, Vance Lemmon, Yunjiao Zhu, Kirill A. Lyapichev, John R. Bethea, Y. Zhang, J. Zha, Nicole M. Ferraro, Jae K. Lee, Stephanie L. Yahn, Dario Motti
Publikováno v:
The Journal of Neuroscience. 37:2362-2376
Although infiltrating macrophages influence many pathological processes after spinal cord injury (SCI), the intrinsic molecular mechanisms that regulate their function are poorly understood. A major hurdle has been dissecting macrophage-specific func
Publikováno v:
Neurobiology of Disease, Vol 74, Iss, Pp 114-125 (2015)
Spinal cord injury (SCI) leads to formation of a fibrotic scar that is inhibitory to axon regeneration. Recent evidence indicates that the fibrotic scar is formed by perivascular fibroblasts, but the mechanism by which they are recruited to the injur
Autor:
José Geraldo Lopes Ramos, Pantelis Tsoulfas, Jae K. Lee, Ezra Blumenthal, Xueting Luo, Catherine Lai-Hsu, Kirill A. Lyapichev, Cynthia Soderblom, Eric R. Bray, Kevin K. Park, Vidhya Krishnan
Publikováno v:
The Journal of Neuroscience. 33:13882-13887
Injury to the CNS leads to formation of scar tissue, which is important in sealing the lesion and inhibiting axon regeneration. The fibrotic scar that comprises a dense extracellular matrix is thought to originate from meningeal cells surrounding the
Autor:
Olga Vorontsova, Jenny E. Hinshaw, Anna Sundborger, Cynthia Soderblom, Oleg Shupliakov, Emma Evergren
Publikováno v:
Journal of Cell Science. 124:133-143
Clathrin-mediated vesicle recycling in synapses is maintained by a unique set of endocytic proteins and interactions. We show that endophilin localizes in the vesicle pool at rest and in spirals at the necks of clathrin-coated pits (CCPs) during acti
Autor:
Michael C. Hanna, Henri J. Jupille, Cynthia Soderblom, Oleg Shupliakov, Julia Stadler, Craig Blackstone
Publikováno v:
neurogenetics. 11:369-378
Mast syndrome (SPG21) is a childhood-onset, autosomal recessive, complicated form of hereditary spastic paraplegia (HSP) characterized by dementia, thin corpus callosum, white matter abnormalities, and cerebellar and extrapyramidal signs in addition
Autor:
Annie Levert, Patrick A. Dion, Cynthia Soderblom, Craig Blackstone, Guy A. Rouleau, Nicolas Dupré, Inge A. Meijer, Peng-Peng Zhu, Sandra Laurent, Michel Sylvain, Jacques Puymirat, Bernard Brais, Julia Stadler
Publikováno v:
Annals of Neurology. 61:599-603
Hereditary spastic paraplegias (HSPs) are characterized by progressive lower limb spasticity and weakness. Mutations in the SPG3A gene, which encodes the large guanosine triphosphatase atlastin, are the second most common cause of autosomal dominant
Autor:
Cynthia Soderblom, Craig Blackstone
Publikováno v:
Pharmacology & Therapeutics. 109:42-56
The hereditary spastic paraplegias (HSPs) comprise a clinically and genetically diverse group of inherited neurological disorders in which the primary manifestation is progressive spasticity and weakness of the lower limbs. The identification of over
Publikováno v:
Journal of neurotrauma. 32(15)
After spinal cord injury (SCI), a fibrotic scar forms at the injury site that is best characterized by the accumulation of perivascular fibroblasts and deposition of the extracellular matrix protein fibronectin. While fibronectin is a growth-permissi
Publikováno v:
Human molecular genetics. 17(11)
The hereditary spastic paraplegias (SPG1-33) comprise a cluster of inherited neurological disorders characterized principally by lower extremity spasticity and weakness due to a length-dependent, retrograde axonopathy of corticospinal motor neurons.
Autor:
Inge A, Meijer, Patrick, Dion, Sandra, Laurent, Nicolas, Dupré, Bernard, Brais, Annie, Levert, Jacques, Puymirat, Marie France, Rioux, Michel, Sylvain, Peng-Peng, Zhu, Cynthia, Soderblom, Julia, Stadler, Craig, Blackstone, Guy A, Rouleau
Publikováno v:
Annals of neurology. 61(6)
Hereditary spastic paraplegias (HSPs) are characterized by progressive lower limb spasticity and weakness. Mutations in the SPG3A gene, which encodes the large guanosine triphosphatase atlastin, are the second most common cause of autosomal dominant