Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Cristina Porras-González"'
Autor:
José Palacios-García, Cristina Porras-González, Ramón Moreno-Luna, Juan Maza-Solano, Juan Polo-Padillo, José Luis Muñoz-Bravo, Serafín Sánchez-Gómez
Publikováno v:
Journal of Clinical Medicine, Vol 12, Iss 9, p 3280 (2023)
Chronic rhinosinusitis with nasal polyps (CRSwNP) is an inflammatory disease of the nose and paranasal sinuses characterized by the presence of nasal polyps. The symptoms produced by the presence of nasal polyps such as nasal obstruction, nasal disch
Externí odkaz:
https://doaj.org/article/f97b0e96b2044dcfb1f9c3ad5b1ff398
Autor:
María del Carmen González-Montelongo, Cristina Porras-González, Rafaela González-Montelongo, Gonzalo Revilla-González, María Dolores Pastor, Antonio Castellano, Juan Ureña
Publikováno v:
Cellular Physiology and Biochemistry, Vol 52, Iss 1, Pp 76-93 (2019)
Externí odkaz:
https://doaj.org/article/8b483ff5de204a609dee709278b9898f
Autor:
Soledad López-Enríquez, Cristina Porras-González, Ramón Moreno-Luna, Charles Stephan Ebert, Gonzalo Alba, Consuelo Santa-María, José María Palacios-García, Juan Maza-Solano, Daniel Iván Martín-Jiménez, Víctor Sánchez-Margalet, Serafín Sánchez-Gómez
Publikováno v:
Archivos de Bronconeumología. 59:337-340
Publikováno v:
Hypertension Research. 41:730-737
Evidence has shown that vascular smooth muscle cells (VSMCs) of spontaneously hypertensive rats (SHRs) are depolarized and that the expression of L-type Ca2+ channels (LTCCs) and the sarcoplasmic reticulum (SR) Ca2+ buffering system are upregulated.
Publikováno v:
Vascular Pharmacology. :33-41
The role of L-type Ca2+ channels (LTCCs) and RhoA/Rho kinase (ROCK) on depolarization-induced sustained arterial contraction lasting several minutes is already known. However, in vivo, vascular smooth muscle cells can be depolarized for longer period
Publikováno v:
Hypertension research : official journal of the Japanese Society of Hypertension. 41(9)
Evidence has shown that vascular smooth muscle cells (VSMCs) of spontaneously hypertensive rats (SHRs) are depolarized and that the expression of L-type Ca
Autor:
Gonzalo Revilla-Gonzalez, Juan Ureña, María del Carmen González-Montelongo, Maria Dolores Pastor, Rafaela González-Montelongo, Cristina Porras-González, Antonio Castellano
Publikováno v:
idUS: Depósito de Investigación de la Universidad de Sevilla
Universidad de Sevilla (US)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Cellular Physiology and Biochemistry, Vol 52, Iss 1, Pp 76-93 (2019)
idUS. Depósito de Investigación de la Universidad de Sevilla
Universidad de Sevilla (US)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Cellular Physiology and Biochemistry, Vol 52, Iss 1, Pp 76-93 (2019)
idUS. Depósito de Investigación de la Universidad de Sevilla
[Background/Aims] Protein kinase C (PKC)- and RhoA/Rho-associated kinase (ROCK) play important roles in arterial sustained contraction. Although depolarization-elicited RhoA/ROCK activation is accepted, the role of PKC in depolarized vascular smooth
Publikováno v:
Vascular pharmacology.
The role of L-type Ca
Autor:
Juan José Egea-Guerrero, María del Carmen González-Montelongo, Juan Ureña, Elena Gordillo-Escobar, Cristina Porras-González, Francisco Murillo-Cabezas, M.A. Muñoz-Sánchez
Publikováno v:
Journal of Physiology and Biochemistry. 70:193-199
The effects of human urotensin II (hUII) on the vascular tone of different animal species has been studied extensively. However, little has been reported on the vasoactive effects of rat urotensin (rUII) in murine models. The aim of the present study
Autor:
Cristina Porras-González, Juan Ureña, María del Carmen González-Montelongo, Francisco Murillo-Cabezas, Ángel Vilches-Arenas, Juan José Egea-Guerrero, M.A. Muñoz-Sánchez, Antonio Castellano
Publikováno v:
Vascular pharmacology. 72
We have previously described that L-type Ca(2+) channels' (LTCCs) activation and metabotropic Ca(2+) release from the sarcoplasmic reticulum (SR) regulate RhoA/Rho kinase (ROCK) activity and sustained arterial contraction. We have investigated whethe