Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Cristina Matthewman"'
Autor:
Tyne W Miller-Fleming, Sarah C Petersen, Laura Manning, Cristina Matthewman, Megan Gornet, Allison Beers, Sayaka Hori, Shohei Mitani, Laura Bianchi, Janet Richmond, David M Miller III
Publikováno v:
eLife, Vol 5 (2016)
Genetic programming and neural activity drive synaptic remodeling in developing neural circuits, but the molecular components that link these pathways are poorly understood. Here we show that the C. elegans Degenerin/Epithelial Sodium Channel (DEG/EN
Externí odkaz:
https://doaj.org/article/9606ca52f9db4fabbe37a8af4fc0f2cb
Publikováno v:
Nutrition Research Reviews. :1-18
The golden spice turmeric with its main bioactive component curcumin is one of the most popular and extensively studied nutraceuticals. Despite numerous pre-clinical studies reporting positive pharmacodynamics of turmeric extracts and curcumin, the m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ef020e1f535082acdc9be4ccbaec9e6e
https://doi.org/10.1017/s0954422423000033
https://doi.org/10.1017/s0954422423000033
Publikováno v:
Molecular Aspects of Medicine. 91:101115
The human gut microbiome is a complex and dynamic microbial entity that interacts with the environment and other parts of the body including the brain, heart, liver, and immune system. These multisystem interactions are highly conserved from inverteb
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c11016d4ab5448639146c8b28de50d10
https://doi.org/10.1016/j.mam.2022.101115
https://doi.org/10.1016/j.mam.2022.101115
Autor:
Andrew G Swick, Kannan R, Balu Maliakel, Aman Khanna, Cristina Matthewman, Krishnakumar Im, Sibi P Ittiyavirah, Syam Das S
Publikováno v:
Nutritional neuroscience. 25(6)
A novel highly bioavailable curcumin-galactomannan (CGM) formulation was shown to have improved blood-brain-barrier (BBB) permeability of free curcuminoids in animal models; however, this has not been established in humans. The present study was cond
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ec737039820dd81cbddc51477054116
https://pubmed.ncbi.nlm.nih.gov/33295851
https://pubmed.ncbi.nlm.nih.gov/33295851
Autor:
John N. Barrett, Cristina Matthewman, Samantha Rincon, Ellen F. Barrett, Suhrud M. Rajguru, Julio Pasos, Jayanti Singh
Publikováno v:
Journal of Neurophysiology. 120:509-524
Inner ear spiral ganglion neurons were cultured from day 4 postnatal mice and loaded with a fluorescent Ca2+indicator (fluo-4, -5F, or -5N). Pulses of infrared radiation (IR; 1,863 nm, 200 µs, 200–250 Hz for 2–5 s, delivered via an optical fiber
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e8f9f5d5f26d76edaeea17859ad7d6d9
https://doi.org/10.1152/jn.00740.2017
https://doi.org/10.1152/jn.00740.2017
Publikováno v:
American Journal of Physiology-Cell Physiology. 315:C155-C163
UNC-8 and MEC-4 are two members of the degenerin/epithelial Na+channel (DEG/ENaC) family of voltage-independent Na+channels that share a high degree of sequence homology and functional similarity. For example, both can be hyperactivated by genetic mu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9df8fd0671abc42f460f8f4b088220ce
https://doi.org/10.1152/ajpcell.00297.2017
https://doi.org/10.1152/ajpcell.00297.2017
Publikováno v:
American Journal of Physiology-Cell Physiology. 311:C920-C930
Hyperactivated DEG/ENaC channels cause neuronal death mediated by intracellular Ca2+overload. Mammalian ASIC1a channels and MEC-4(d) neurotoxic channels in Caenorhabditis elegans both conduct Na+and Ca2+, raising the possibility that direct Ca2+influ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ba60ff6599eef64d0a276c8fe1a948d
https://doi.org/10.1152/ajpcell.00247.2016
https://doi.org/10.1152/ajpcell.00247.2016
Publikováno v:
The Journal of General Physiology
Ion channels of the DEG/ENaC family can induce neurodegeneration under conditions in which they become hyperactivated. The Caenorhabditis elegans DEG/ENaC channel MEC-4(d) encodes a mutant channel with a substitution in the pore domain that causes sw
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7682b5656eb8a1c789aa2f5eed35a360
https://doi.org/10.1085/jgp.201310974
https://doi.org/10.1085/jgp.201310974
Autor:
David M. Miller, Allison Beers, Sayaka Hori, Sarah C. Petersen, Shohei Mitani, Janet E. Richmond, Tyne W. Miller-Fleming, Laura Bianchi, Laura Manning, Megan Gornet, Cristina Matthewman
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2378b2d79280ffb598106663465713a9
https://doi.org/10.7554/elife.14599.021
https://doi.org/10.7554/elife.14599.021
