Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Cristina Lao-Peregrín"'
Autor:
Xavier Altafaj, Eduardo D. Martín, Jon Ortiz-Abalia, Aitana Valderrama, Cristina Lao-Peregrín, Mara Dierssen, Cristina Fillat
Publikováno v:
Neurobiology of Disease, Vol 52, Iss , Pp 117-127 (2013)
The cognitive dysfunctions of Down Syndrome (DS) individuals are the most disabling alterations caused by the trisomy of human chromosome 21 (HSA21). In trisomic Ts65Dn mice, a genetic model for DS, the overexpression of HSA21 homologous genes has be
Externí odkaz:
https://doaj.org/article/708a6ae4ef8d4bb9b8c8cf2afa7fc8ac
Autor:
Cristina Lao-Peregrin, Guoqing Xiang, Jihye Kim, Ipsit Srivastava, Alexandra B. Fall, Danielle M. Gerhard, Piia Kohtala, Daegeon Kim, Minseok Song, Mikel Garcia-Marcos, Joshua Levitz, Francis S. Lee
Publikováno v:
Cell Reports, Vol 43, Iss 1, Pp 113595- (2024)
Summary: Cellular signaling involves a large repertoire of membrane receptors operating in overlapping spatiotemporal regimes and targeting many common intracellular effectors. However, both the molecular mechanisms and the physiological roles of cro
Externí odkaz:
https://doaj.org/article/28edfc249a87468a91385cfb259ade66
Autor:
Esther Pérez-Navarro, María Gómez Lázaro, Deborah J. Burks, Cristina Lao-Peregrín, Miriam Fernández, Ana Saavedra, Alfonsa Zamora-Moratalla, Eduardo D. Martín, Jesús J. Ballesteros
Publikováno v:
Addiction Biology. 22:1706-1718
Caffeine has cognitive-enhancing properties with effects on learning and memory, concentration, arousal and mood. These effects imply changes at circuital and synaptic level, but the mechanism by which caffeine modifies synaptic plasticity remains el
Autor:
Aitana Valderrama, Xavier Altafaj, Eduardo D. Martín, Mara Dierssen, Cristina Lao-Peregrín, Jon Ortiz-Abalia, Cristina Fillat
Publikováno v:
Neurobiology of Disease, Vol 52, Iss, Pp 117-127 (2013)
The cognitive dysfunctions of Down Syndrome (DS) individuals are the most disabling alterations caused by the trisomy of human chromosome 21 (HSA21). In trisomic Ts65Dn mice, a genetic model for DS, the overexpression of HSA21 homologous genes has be
Publikováno v:
Epilepsia. 51:384-390
Summary Purpose: In this study, we explore the antiepileptic effects of flufenamic acid (FFA) in order to identify the cellular mechanisms that underlie the potential anticonvulsant properties of this nonsteroidal antiinflammatory compound. Methods: