Zobrazeno 1 - 10 of 14 pro vyhledávání: '"Cristhine Pastorini"'
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Akademický článek
TLR4 activates the β-catenin pathway to cause intestinal neoplasia.
Autor: Rebeca Santaolalla, Daniel A Sussman, Jose R Ruiz, Julie M Davies, Cristhine Pastorini, Cecilia L España, John Sotolongo, Oname Burlingame, Pablo A Bejarano, Sakhi Philip, Mansoor M Ahmed, Jeffrey Ko, Ramanarao Dirisina, Terrence A Barrett, Limin Shang, Sergio A Lira, Masayuki Fukata, Maria T Abreu
Publikováno v: PLoS ONE, Vol 8, Iss 5, p e63298 (2013)
Colonic bacteria have been implicated in the development of colon cancer. We have previously demonstrated that toll-like receptor 4 (TLR4), the receptor for bacterial lipopolysaccharide (LPS), is over-expressed in humans with colitis-associated cance
Externí odkaz: https://doaj.org/article/9a0ce1c7df5e4b148977f947a7b0267a
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2
Intestinal Epithelial Toll-Like Receptor 4 Signaling Affects Epithelial Function and Colonic Microbiota and Promotes a Risk for Transmissible Colitis
Autor: Rebeca Santaolalla, Maria T. Abreu, Cristhine Pastorini, Maria T. Vazquez-Pertejo, Matthew C. Phillips, Jessica K. Lang, Rishu Dheer, Julie M. Davies
Publikováno v: Infection and Immunity
Evidence obtained from gene knockout studies supports the role of Toll-like receptor 4 (TLR4) in intestinal inflammation and microbiota recognition. Increased epithelial TLR4 expression is observed in patients with inflammatory bowel disease. However
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c9e54d77301a484381ee769374be42ef
https://doi.org/10.1128/iai.01374-15
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4
The role of prostaglandin E2 (PGE 2) in toll-like receptor 4 (TLR4)-mediated colitis-associated neoplasia
Autor: Cristhine Pastorini, Anli Chen, Daisy E. Conduah, Keith J. Breglio, Maria T. Abreu, Ruliang Xu, John P. Sotolongo, David Hsu, Lory Hayes, Yasmín Hernández, Ryan C. Ungaro, Masayuki Fukata
Publikováno v: BMC Gastroenterology, Vol 10, Iss 1, p 82 (2010)
BMC Gastroenterology
BackgroundWe have previously found that TLR4-deficient (TLR4-/-) mice demonstrate decreased expression of mucosal PGE2and are protected against colitis-associated neoplasia. However, it is still unclear whether PGE2is the central factor downstream of
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3913037a1c9b2aa3b19da265e4ddc27
https://doi.org/10.1186/1471-230x-10-82
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5
TLR4 Activates the β-catenin Pathway to Cause Intestinal Neoplasia
Autor: Maria T. Abreu, Jeffrey Z. Ko, Daniel A. Sussman, Julie M. Davies, Oname Burlingame, Pablo A. Bejarano, Limin Shang, Mansoor M. Ahmed, Jose R. Ruiz, John P. Sotolongo, Sergio A. Lira, Rebeca Santaolalla, Sakhi Philip, Terrence A. Barrett, Cecilia Espana, Ramanarao Dirisina, Masayuki Fukata, Cristhine Pastorini
Publikováno v: PLoS ONE, Vol 8, Iss 5, p e63298 (2013)
PLoS ONE
Colonic bacteria have been implicated in the development of colon cancer. We have previously demonstrated that toll-like receptor 4 (TLR4), the receptor for bacterial lipopolysaccharide (LPS), is over-expressed in humans with colitis-associated cance
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29d3309f1384d0a004420a8c3d14b82e
https://doi.org/10.1371/journal.pone.0063298
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