Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Craig S. Hill"'
Publikováno v:
Journal of Great Lakes Research. 48:478-488
Autor:
Daniel Ilacas, John R. Farley, Christopher Orcutt, Barbara E. Miller, Craig S. Hill, David J. Baylink, Susan L. Hall
Publikováno v:
Clinical Chemistry. 40:1749-1756
Three methods for quantifying skeletal alkaline phosphatase (ALP; EC 3.1.3.1) activity/immunoactivity in serum--heat inactivation, wheat germ agglutinin (WGA) precipitation, and an immunoradiometric assay--were tested for recovery and specificity and
Autor:
Florence, Paillard, Craig S, Hill
Publikováno v:
MLO: medical laboratory observer. 36(1)
Partnering with a reference lab can actually help grow a referring lab's own business. The reference lab provides a hospital's physicians with access to unique tests and clinical information and broadens the scope and depth of the referring lab's men
Autor:
Craig S. Hill, Larry F. Lemanski
Publikováno v:
Journal of Experimental Zoology. 209:1-19
Recessive mutant gene c in Ambystoma mexicanum results in an absence of embryonic heart function. When mutant hearts are transplanted into normal embryos, the defect is corrected and the mutant hearts beat. In reciprocal transplants normal hearts do
Immunoelectron microscopic localization of alpha-actinin on Lowicryl-embedded thin-sectioned tissues
Publikováno v:
The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society. 33(6)
A procedure has been developed for the immunoelectron microscopic localization of intracellular antigens on thin-sectioned tissues. The tissues were fixed in a periodate-lysine-paraformaldehyde solution or a formaldehyde-glutaraldehyde combination an
Publikováno v:
Science (New York, N.Y.). 196(4292)
Homozygosity for simple recessive gene c in axolotl embryos results in the absence of a heartbeat. Gene c alters the morphology of the mutant anterior endoderm - the primary heart inductor.
Publikováno v:
Science (New York, N.Y.). 204(4395)
Recessive mutant gene c in axolotl embryos results in an absence of heart function. Normal (+/+) anterior endoderm cultured with mutant (c/c) hearts totally corrects the defect.