Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Craig, Ricci"'
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 86:166-172
Glucose toxicity is an important initiator of cardiovascular disease, contributing to the development of insulin resistance, impaired contractile function, abnormal energy metabolism, cardiomyocyte and endothelial cell death, coronary heart disease,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a393be39bbe3fcf16bdaa16be2d664d9
https://doi.org/10.1139/y08-021
https://doi.org/10.1139/y08-021
Autor:
Julio F. Turrens, Viktor Pastukh, David V. Schaffer, Stephen W. Schaffer, Glenn L. Wilson, Josh Leonard, Craig Ricci
Publikováno v:
American Journal of Physiology-Cell Physiology. 294:C413-C422
Recently, it has become apparent that mitochondrial DNA (mtDNA) damage can rapidly initiate apoptosis independent of mutations, although the mechanism involved remains unclear. To elucidate this mechanism, angiotensin II-mediated apoptosis was studie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53ee94b895b39cffc73c4202f3dfc9dc
https://doi.org/10.1152/ajpcell.00362.2007
https://doi.org/10.1152/ajpcell.00362.2007
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 85:455-464
Diabetes is characterized by chronic hyperglycemia as well as insulin deficiency or resistance. However, the majority of research has focused on the consequences of hyperglycemia in development of diabetic complications, whereas the effects of insuli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::feba3a042906358238e703b63518e9c9
https://doi.org/10.1139/y07-029
https://doi.org/10.1139/y07-029
Publikováno v:
Experimental and clinical cardiology. 10(3)
Angiotensin II plays a central role in the development of congestive heart failure. Among its many actions linked to heart failure is the promotion of ventricular remodelling. Although the growth component of ventricular remodelling may initially be
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::f26e86d3aa9218430157f44e16ac7d62
https://pubmed.ncbi.nlm.nih.gov/19641681
https://pubmed.ncbi.nlm.nih.gov/19641681
Publikováno v:
Canadian journal of physiology and pharmacology. 86(4)
Glucose toxicity is an important initiator of cardiovascular disease, contributing to the development of insulin resistance, impaired contractile function, abnormal energy metabolism, cardiomyocyte and endothelial cell death, coronary heart disease,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::fe98ac2f6c8803245385230490c31655
https://pubmed.ncbi.nlm.nih.gov/18418425
https://pubmed.ncbi.nlm.nih.gov/18418425
Publikováno v:
The FASEB Journal. 22
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1f3f7bc832ee5f7c19bb3e7c499f48b1
https://doi.org/10.1096/fasebj.22.1_supplement.751.16
https://doi.org/10.1096/fasebj.22.1_supplement.751.16
Publikováno v:
The FASEB Journal. 20
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::14aa895a366a706a26432383c41b5da5
https://doi.org/10.1096/fasebj.20.4.a121-b
https://doi.org/10.1096/fasebj.20.4.a121-b
Publikováno v:
Molecular and cellular biochemistry. 269(1-2)
Osmolytes are rapidly lost from the ischemic heart, an effect thought to benefit the heart by reducing the osmotic load. However, the observation that chronic lowering of one of the prominent osmolytes, taurine, is more beneficial to the ischemic hea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8dfe48757f93d40e0cdde1a6f2142a8
https://pubmed.ncbi.nlm.nih.gov/15786717
https://pubmed.ncbi.nlm.nih.gov/15786717
Publikováno v:
Journal of Molecular and Cellular Cardiology. 40:896
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5af63d6f4b95a392b90354c4c4d6de84
https://doi.org/10.1016/j.yjmcc.2006.03.374
https://doi.org/10.1016/j.yjmcc.2006.03.374
Publikováno v:
Molecular & Cellular Biochemistry; Jan2005, Vol. 269 Issue 1, p59-67, 9p