Výsledky vyhledávání - "Costet, Philippe"

Akademický článek

PCSK9: a promising therapeutic target for dyslipidemias?

Autor: Lambert, Gilles, Krempf, Michel, Costet, Philippe

Publikováno v: In Trends in Endocrinology & Metabolism 2006 17(3):79-81

Akademický článek

Plasma PCSK9 concentrations during an oral fat load and after short term high-fat, high-fat high-protein and high-fructose diets

Autor: Cariou Bertrand, Langhi Cédric, Le Bras Maëlle, Bortolotti Murielle, Lê Kim-Anne, Theytaz Fanny, Le May Cédric, Guyomarc’h-Delasalle Béatrice, Zaïr Yassine, Kreis Roland, Boesch Chris, Krempf Michel, Tappy Luc, Costet Philippe

Publikováno v: Nutrition & Metabolism, Vol 10, Iss 1, p 4 (2013)

Abstract Background PCSK9 (Proprotein Convertase Subtilisin Kexin type 9) is a circulating protein that promotes hypercholesterolemia by decreasing hepatic LDL receptor protein. Under non interventional conditions, its expression is driven by sterol

Externí odkaz: https://doaj.org/article/41228e392acb4bd3a535e01ac8e20f2c

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PCSK9 dominant negative mutant results in increased LDL catabolic rate and familial hypobetalipoproteinemia

Autor: Le May, Cedric, Cariou, Bertrand, Ouguerram, Khadija, Zaïr, Yassine, Guerois, Raphael, Langhi, Cédric, Kourimate, Sanae, Benoit, Isabelle, Le May, Cédric, Gayet, Constance, Belabbas, Khaldia, Dufernez, Fabienne, Chétiveaux, Maud, Tarugi, Patrizia, Krempf, Michel, Benlian, Pascale, Costet, Philippe

Publikováno v: Arteriosclerosis, Thrombosis, and Vascular Biology
Arteriosclerosis, Thrombosis, and Vascular Biology, American Heart Association, 2009, 29 (12), pp.2191-2197. ⟨10.1161/ATVBAHA.109.194191⟩

Objective— Proprotein convertase subtilisin/kexin type 9 (PCSK9) is a central player in the regulation of cholesterol homeostasis, increasing the low-density lipoprotein (LDL) receptor degradation. Our study aimed at exploring the pathogenic conseq

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ce3fa0d5d00a31cedbd36308892384cc
https://hdl.handle.net/11380/638383

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