Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Consuelo Ventura-Mejía"'
Publikováno v:
Acta Neurobiologiae Experimentalis.
Aminopyridines constitute a drug family with the ability to enhance synaptic transmission. In particular, 4‑aminopyridine (4‑AP) has been used as a model of generalized seizures. 4‑AP is a K+ channel blocker, but its mechanism of action has not
Autor:
Consuelo Ventura-Mejía, Sofía Rodríguez-Mercado, Fridha Villalpando-Vargas, Laura Medina-Ceja
Publikováno v:
e-CUCBA. :8-17
Autor:
Laura Guadalupe Medina Ceja, Fridha Viridiana Villalpando Vargas, Consuelo Ventura Mejía, Sofia Rodríguez Mercado
Publikováno v:
e-CUCBA. :54-68
Publikováno v:
e-CUCBA.
Publikováno v:
NeuroImage. 113:374-386
Status epilepticus is an emergency condition in patients with prolonged seizure or recurrent seizures without full recovery between them. The pathophysiological mechanisms of status epilepticus are not well established. With this argument, we use a c
Publikováno v:
BioMed Research International, Vol 2014 (2014)
BioMed Research International
BioMed Research International
Background. In models of temporal lobe epilepsy and in patients with this pathology, high frequency oscillations called fast ripples (FRs, 250–600 Hz) can be observed. FRs are considered potential biomarkers for epilepsy and, in the light of manyin
Autor:
Kenia Pardo-Peña, Consuelo Ventura-Mejía, Armando López-Cuevas, B. Castillo-Toledo, Laura Medina-Ceja
Publikováno v:
Computer Methods and Programs in Biomedicine. 110:354-360
Recent studies suggest that the appearance of signals with high frequency oscillations components in specific regions of the brain is related to the incidence of epilepsy. These oscillations are in general small in amplitude and short in duration, ma
Publikováno v:
Neuroreport. 25(11)
The pilocarpine model of temporal lobe epilepsy (TLE) is a useful tool that is used to investigate the mechanisms underlying the generation and maintenance of seizures. Although this model has been modified significantly to reduce mortality and to pr
Publikováno v:
Seizure. 19(8)
In vivo and in vitro evidence from animals suggesting that gap junctions (GJs) play a role in the spreading of epileptiform activity. We have examined the influence of the gap junction opener trimethylamine (TMA) and the connexin 36 (Cx36) gap juncti