Abstract WP101: Vessel Wall Enhancement Is Associated With Symptomatic Adult Moyamoya Disease And Syndrome In An American Population

Autor: Conny H Tran, Chahat Rana, William Moore, Ty T Shang

Publikováno v: Stroke. 54

Introduction: Moyamoya Disease (MMD) and Moyamoya Syndrome (MMS) are chronic progressive cerebrovascular disease characterized by bilateral or unilateral stenotic arteries around the circle of Willis with subsequent development of collateral blood ve

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::762a20a96c29fad5662ce755a8fc83aa
https://doi.org/10.1161/str.54.suppl_1.wp101

Pathological high frequency oscillations associate with increased GABA synaptic activity in pediatric epilepsy surgery patients

Autor: Harry V. Vinters, Carlos Cepeda, Vannah Wila Yazon, Aria Fallah, Conny H Tran, Joshua Barry, Hiroki Nariai, Simon Levinson, Katerina D. Oikonomou, Gary W. Mathern, Joyce Y. Wu

Publikováno v: Neurobiology of Disease, Vol 134, Iss, Pp-(2020)
Neurobiol Dis

Pathological high-frequency oscillations (HFOs), specifically fast ripples (FRs, >250 Hz), are pathognomonic of an active epileptogenic zone. However, the origin of FRs remains unknown. Here we explored the correlation between FRs recorded with intra

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9d8d75008daac6b0d34c2317d351a62d
http://www.sciencedirect.com/science/article/pii/S0969996119302931

Altered excitatory and inhibitory inputs to striatal medium-sized spiny neurons and cortical pyramidal neurons in the Q175 mouse model of Huntington's disease

Autor: Michael Levine, Tim Indersmitten, Carlos Cepeda, Conny H Tran

Publikováno v: Journal of Neurophysiology. 113:2953-2966

The Q175 knockin mouse model of Huntington's disease (HD) carries a CAG trinucleotide expansion of the human mutant huntingtin allele in its native mouse genomic context and recapitulates the genotype more closely than transgenic models. In this stud

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1d8cba9761e31395da55ecb3da89a00
https://doi.org/10.1152/jn.01056.2014

In Rasmussen Encephalitis, Hemichannels Associated with Microglial Activation are linked to Cortical Pyramidal Neuron Coupling: A Possible Mechanism for Cellular Hyperexcitability

Autor: Carlos Cepeda, Jane Y. Chen, Michael S. Levine, Harry V. Vinters, My N. Huynh, Gary W. Mathern, Geoffrey C. Owens, Julia W. Chang, Conny H Tran

Publikováno v: Cepeda, C; Chang, JW; Owens, GC; Huynh, MN; Chen, JY; Tran, C; et al.(2014). In Rasmussen Encephalitis, Hemichannels Associated with Microglial Activation are linked to Cortical Pyramidal Neuron Coupling: A Possible Mechanism for Cellular Hyperexcitability. CNS Neuroscience and Therapeutics. doi: 10.1111/cns.12352. UCLA: Retrieved from: http://www.escholarship.org/uc/item/57f1d2fg
CNS neuroscience & therapeutics, vol 21, iss 2
CNS Neuroscience and Therapeutics, vol 21, iss 2
Cepeda, C; Chang, JW; Owens, GC; Huynh, MN; Chen, JY; Tran, C; et al.(2015). In rasmussen encephalitis, hemichannels associated with microglial activation are linked to cortical pyramidal neuron coupling: A possible mechanism for cellular hyperexcitability. CNS Neuroscience and Therapeutics, 21(2), 152-163. doi: 10.1111/cns.12352. UCLA: Retrieved from: http://www.escholarship.org/uc/item/5d90t8q8

© 2014 John Wiley & Sons Ltd. Aims: Rasmussen encephalitis (RE) is a rare but devastating condition, mainly in children, characterized by sustained brain inflammation, atrophy of one cerebral hemisphere, epilepsy, and progressive cognitive deteriora

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42843ea5170236302cb507145bdc52f4
http://www.escholarship.org/uc/item/57f1d2fg