Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Conner P Witherow"'
Autor:
Manveen K. Gupta, Anita Sahu, Yu Sun, Maradumane L. Mohan, Avinash Kumar, Ajaykumar Zalavadia, Xi Wang, Elizabeth E. Martelli, Kate Stenson, Conner P. Witherow, Judy Drazba, Srinivasan Dasarathy, Sathyamangla V. Naga Prasad
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-16 (2021)
Abstract Although microRNA-7 (miRNA-7) is known to regulate proliferation of cancer cells by targeting Epidermal growth factor receptor (EGFR/ERBB) family, less is known about its role in cardiac physiology. Transgenic (Tg) mouse with cardiomyocyte-s
Externí odkaz:
https://doaj.org/article/51f839007800465793b14c9b05827ac8
Autor:
Sathyamangla V. Naga Prasad, Robert S. Papay, Yu Sun, Conner P Witherow, Kate Stenson, Maradumane L. Mohan
Publikováno v:
Circulation Research. 129
Genetic deletion of Phosphoinositide 3-kinase (PI3Kγ) in mice (PI3Kγ -/- ) results in increased cAMP levels and enhanced ventricular rate/contractility. We investigated whether PI3Kγ plays a role in cardiac contractility by altering intracellular
Autor:
John Barnard, Conner P Witherow, Mina K. Chung, Sathyamangla V. Naga Prasad, David R. Van Wagoner, Nana Liu, Jonathan D. Smith
Publikováno v:
Circulation. 142
Introduction: SYNE2 encodes a nuclear membrane protein that connects the nucleus with the cytoskeleton. rs1152591 is a common SNP in the SYNE2 gene that is associated with atrial fibrillation (AF) and with reduced expression of SYNE2α1 , a short iso
Autor:
Mohan L Maradumane, Vikram Sharma, Ruchi Sharma, Sathyamangala Prasad, Conner P Witherow, Venu Menon, Wai Hong W Tang, Shehab AlAnsari
Publikováno v:
Circulation. 142
Introduction: Circulating blood troponin complexes and free fractions remain poorly characterised in different conditions where troponin is detectable in blood Hypothesis: The aim of the study was to compare the differences in troponin-I(TnI) complex
Publikováno v:
Circulation Research. 127
Genetic deletion of Phosphoinositide 3-kinase (PI3Kγ) in mice (PI3Kγ -/- ) results in increased cAMP levels and enhanced ventricular contractility. We investigated whether the lack of PI3Kγ plays a role in cardiac contractility by altering intrace